Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells.

K. Hieshima; D. Nagakubo; T. Nakayama; A.-K. Shirakawa; Z. Jin; O. Yoshie

首页> 外文期刊>The journal of immunology >Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells.

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Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells.

机译：人T细胞白血病病毒1型（HTLV-1）感染的T细胞的税收诱导CC趋化因子配体22的产生促进了HTLV-1优先向表达CCR4的CD4 + T细胞的传播。

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Hieshima, K., D. Nagakubo, T. Nakayama, A.-K. Shirakawa, Z. Jin, and O. Yoshie. 2008. Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells. [ J. Immunol. 180:

机译：广岛（Hieshima），长久保D.Nagakubo（中山T.）白河，Z。Jin和O. Yoshie。 2008年。人T细胞白血病病毒1型（HTLV-1）感染的T细胞可诱导税收诱导的CC趋化因子配体22的产生，促进HTLV-1向表达CCR4的CD4 + T细胞的优先传播。 [J. Immunol。 180：

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《The journal of immunology》 |2008年第12期|共1页
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K. Hieshima; D. Nagakubo; T. Nakayama; A.-K. Shirakawa; Z. Jin; O. Yoshie;
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1. Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells. [J] . Hieshima K, Nagakubo D, Nakayama T, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2008,第2期

机译：人T细胞白血病病毒1型（HTLV-1）感染的T细胞的税诱导CC趋化因子配体22的产生促进了HTLV-1优先向表达CCR4的CD4 + T细胞的传播。
2. Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells. [J] . Hieshima K, Nagakubo D, Nakayama T, The Journal of Immunology: Official Journal of the American Association of Immunologists . 2008,第2期

机译：人T细胞白血病病毒1型（HTLV-1）感染的T细胞的税诱导CC趋化因子配体22的产生促进了HTLV-1优先向表达CCR4的CD4 + T细胞的传播。
3. Tax-Inducible Production of CC Chemokine Ligand 22 by Human T Cell Leukemia Virus Type 1 (HTLV-1)-Infected T Cells Promotes Preferential Transmission of HTLV-1 to CCR4-Expressing CD4+ T Cells [J] . Kunio Hieshima, Daisuke Nagakubo, Takashi Nakayama, The journal of immunology . 2008,第2期

机译：由人T细胞白血病病毒1型（HTLV-1）感染的T细胞的税收诱导生产CC趋化因子配体22促进了HTLV-1向表达CCR4的CD4 + T细胞的优先传播。
4. SINGLE-CELL ANALYSIS OF INFLUENZA A VIRUS-INFECTED CELLS FOR THE OPTIMIZATION OF CELL CULTURE-BASED VACCINE PRODUCTION [C] . Sascha Young Kupke, Bioprocess Engineering, Timo Frensing, Vaccine technology VI . 2016

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5. Evaluating the role of dendritic cells in host immune response during human T cell leukemia virus type 1 (HTLV-1) infection using a transgenic mouse model system [D] . Rahman, Saifur 2010

机译：使用转基因小鼠模型系统评估树突状细胞在人T细胞白血病病毒1型（HTLV-1）感染期间宿主免疫反应中的作用
6. The Tax-Inducible Actin-Bundling Protein Fascin Is Crucial for Release and Cell-to-Cell Transmission of Human T-Cell Leukemia Virus Type 1 (HTLV-1) [O] . Christine Gross, Veit Wiesmann, Sebastian Millen, 2016

机译：税收诱导肌动蛋白捆绑蛋白Fascin对于人类T细胞白血病病毒1型（HTLV-1）的释放和细胞间传播至关重要。
7. T CD4+ cells count among patients co-infected with human immunodeficiency virus type 1 (HIV-1) and human T-cell leukemia virus type 1 (HTLV-1): high prevalence of tropical spastic paraparesis/HTLV-1-associated myelopathy (TSP/HAM) Contagens de células T CD4+ na co-infecção HIV-1 e HTLV-1: alta prevalência da paraparesia espástica tropical/mielopatia associada ao HTLV-1 [O] . Jorge Casseb, Maria Paulina Posada-Vergara, Patrícia Montanheiro, 2007

机译：共感染1型人类免疫缺陷病毒（HIV-1）和人类T细胞白血病病毒1型（HTLV-1）的患者中T CD4 +细胞计数：热带痉挛性下肢瘫痪/ HTLV-1相关性脊髓病（Tsp）患病率高/ Ham）ContagensdecélulasTCD4 + na co-infecçãoHIV-1 e HTLV-1：altaprevalênciadaparaparesiaespásticactropical/ mielopatia associada ao HTLV-1

Tax-inducible production of CC chemokine ligand 22 by human T cell leukemia virus type 1 (HTLV-1)-infected T cells promotes preferential transmission of HTLV-1 to CCR4-expressing CD4+ T cells.

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