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首页> 外文期刊>Frontiers in Psychology >Training, hypnosis, and drugs: artificial synaesthesia, or artificial paradises?
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Training, hypnosis, and drugs: artificial synaesthesia, or artificial paradises?

机译:训练,催眠和药物:人工通感还是人工天堂?

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The last few years have seen the publication of a number of studies by researchers claiming to have induced “synaesthesia,” “pseudo-synaesthesia,” or “synaesthesia-like” phenomena in non-synaesthetic participants. Although the intention of these studies has been to try and shed light on the way in which synaesthesia might have been acquired in developmental synaesthestes, we argue that they may only have documented a phenomenon that has elsewhere been accounted for in terms of the acquisition of sensory associations and is not evidently linked to synaesthesia. As synaesthesia remains largely defined in terms of the involuntary elicitation of conscious concurrents, we suggest that the theoretical rapprochement with synaesthesia (in any of its guises) is unnecessary, and potentially distracting. It might therefore, be less confusing if researchers were to avoid referring to synaesthesia when characterizing cases that lack robust evidence of a conscious manifestation. Even in the case of those other conditions for which conscious experiences are better evidenced, when training has been occurred during hypnotic suggestion, or when it has been combined with drugs, we argue that not every conscious manifestation should necessarily be counted as synaesthetic. Finally, we stress that cases of associative learning are unlikely to shed light on two highly specific characteristic of the majority of cases of developmental synaesthesia in terms of learning patterns: First, their resistance to change through exposure once the synaesthetic repertoire has been fixed; Second, the transfer of conditioned responses between concurrents and inducers after training. We conclude by questioning whether, in adulthood, it is ever possible to acquire the kind of synaesthesia that is typically documented in the developmental form of the condition. The available evidence instead seems to point to there being a critical period for the development of synaesthesia, probably only in those with a genetic predisposition to develop the condition.
机译:最近几年,研究人员发表了许多研究,这些研究声称在非麻醉参与者中诱发了“感觉异常”,“假感觉异常”或“类似感觉异常”的现象。尽管这些研究的目的是试图阐明发育联觉中通感的获得方式,但我们认为它们可能仅记录了一种现象,该现象在感觉获得方面已被其他人解释。协会,并没有明显联系联觉。由于通感仍在很大程度上由有意识的并发的非自愿诱发来定义,因此我们建议与通感(在任何形式上)的理论上的和解是不必要的,并且可能分散注意力。因此,如果研究人员在表征缺乏有力表现的有力证据的病例时避免使用联觉,可能会减少混乱。即使在其他条件下,有更好的自觉体验,在催眠提示期间进行了训练,或者与药物联合使用时,我们认为并非每个有意识的表现都必须算作联觉。最后,我们强调,在学习模式方面,联想学习案例不太可能阐明大多数发育性联觉案例的两个高度特定的特征:首先,一旦确定了联觉库,他们就不会通过暴露改变。第二,训练后并发与诱导之间条件响应的转移。我们通过质疑在成年后是否有可能获得通常以病情发展形式记录的通感的结论。相反,可用的证据似乎指向联觉发展有一个关键时期,可能只有在具有遗传易感性疾病的人中。

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