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首页> 外文期刊>Polish Archives of Internal Medicine >Complications of permanent dual?chamber pacing such as late purulent pacemaker pocket infection with broken and looped atrial lead, complicated by pulmonary embolism after transvenous lead removal: a case report
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Complications of permanent dual?chamber pacing such as late purulent pacemaker pocket infection with broken and looped atrial lead, complicated by pulmonary embolism after transvenous lead removal: a case report

机译:永久性双腔起搏的并发症,例如晚期化脓性起搏器口袋感染,伴有心房导线断裂和环行,并经静脉移出铅后并发肺栓塞:一例

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We present a complication of the infected pacing system extraction by lobular pneumonia in a 73?year?old female patient. The pacing system involved DDD pacemaker, atrial and ventricular endocardial leads implanted 12 year beforehand. The defect of the atrial lead emerged during the pacemaker replacement 4 years ago. The diagnosis of the injury cause and its reparation were not undertaken at that time. An interruption of the atrial lead which resulted in the formation of a loop inside the cardiac chamber was found when purulant pacemaker pocket infection had been diagnosed. The patient was referred for the pacing system extraction after preoperative specific antibiotic treatment. After a long?lasting, difficult, two?step leads extraction procedure, pneumonia developed. An echocardiogram revealed enlargement of the right atrium and ventricle, with elevated pulmonary artery pressure up to 40 mmHg. An atypical chest X?ray with the presence of a large pleural liquid volume led to the work?up of hemorrhagic complications and postponed the antithrombotic therapy. With the delay of 1.5 month the pulmonary scintigraphy showed features of pulmonary embolism. The embolism was most likely caused by a vegetation mobilized from the endocardial lead and/or endocardium during the extraction maneuvers. Before the surgery, the vegetations attached to the leads or to the endocardium had not been visualized. Anticoagulant therapy with antivitamins K was successful, which resulted in the pulmonary pressure normalization. The patient has remained in a good condition for the next 3 months of follow?up.
机译:我们提出了73岁女性患者小叶性肺炎感染起搏系统提取的并发症。起搏系统包括DDD起搏器,心房和心室心内膜导线,这些导线已提前12年植入。 4年前更换心脏起搏器时出现了心房导联的缺陷。当时尚未进行受伤原因的诊断和赔偿。当诊断出化脓性起搏器口袋感染时,发现导致心腔内形成环路的心房导线中断。术前经过特殊抗生素治疗后,将患者转入起搏系统中。经过长时间,困难,两步的引线提取程序后,出现了肺炎。超声心动图显示右心房和心室增大,肺动脉压升高至40 mmHg。不典型的胸部X光片有大量胸腔积液,导致出血并发症的发生,并推迟了抗栓治疗。延迟1.5个月,肺闪烁显像显示出肺栓塞的特征。栓塞很可能是由于在提取操作期间从心内膜导联和/或心内膜动员的植被引起的。手术之前,未观察到附着在导线或心内膜上的植物。抗维生素K的抗凝治疗是成功的,从而使肺压正常化。在接下来的三个月随访中,患者一直处于良好状态。

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