Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau

Stefan Thor; Malgorzata Pokrzywa; Josefin Fernius; Annika Starkenberg

首页> 外文期刊>Biology Open >Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau

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Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau

机译：人类Tau的共表达加剧了果蝇中人TTBK1，TTBK2和MARK1激酶的毒性人类Dau的共表达而加剧了果蝇中人TTBK1，TTBK2和MARK1激酶的毒性加剧了Dau中的人类Tau的人类TTBK1，TTBK2和MARK1激酶毒性人类Tau的共表达加剧了黑色素瘤

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cannot no abstract right now, maybe has no abstract or it's too hard to get

机译：现在不能没有摘要，可能没有摘要，或者太难获取

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《Biology Open》 |2017年第7期|共页
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Stefan Thor; Malgorzata Pokrzywa; Josefin Fernius; Annika Starkenberg;
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入库时间 2022-08-18 06:54:33

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1. Mutant human torsinA, responsible for early-onset dystonia, dominantly suppresses GTPCH expression, dopamine levels and locomotion in Drosophila melanogaster Mutant human torsinA, responsible for early-onset dystonia, dominantly suppresses GTPCH expression, dopamine levels and locomotion in Drosophila melanogaster Mutant human torsinA, responsible for early-onset dystonia, dominantly suppresses GTPCH expression, dopamine levels and locomotion in Drosophila melanogaster [J] . Janis M. ODonnell, Xandra O. Breakefield, Rami R. Ajjuri, Biology Open . 2015,第5期

机译：负责早发性肌张力障碍的突变人类躯体A，主要抑制果蝇的GTPCH表达，多巴胺水平和运动负责人早发性肌张力障碍的突变人类躯体A，主要抑制果蝇的GTPCH表达，多巴胺水平和运动，突变体。负责早期发作的肌张力障碍，主要抑制果蝇的GTPCH表达，多巴胺水平和运动
2. Non-homologous recombination of deoxyribonucleoside kinases from human and Drosophila melanogaster yields human-like enzymes with novel activities [J] . Gerth ML, Lutz S Journal of Molecular Biology . 2007,第4期

机译：来自人和果蝇的脱氧核糖核苷激酶的非同源重组产生具有新颖活性的人样酶
3. Pancreatic eukaryotic initiation factor-2α kinase (PEK) homologues in humans, Drosophila melanogaster and Caenorhabditis elegans that mediate translational control in response to endoplasmic reticulum stress [J] . Amy C. PORTER, Kun MA, Lawrence A. QUILLIAM, The biochemical journal . 2000,第2期

机译：人类，果蝇和秀丽隐杆线虫中的胰腺真核生物起始因子2α激酶（PEK）同源物，它们响应内质网应激介导翻译控制
4. Using Drosophila melanogaster Data to Discover Disease-Related Protein Interactions in Humans [C] . Costello, J.C., Buchanan-Carter, . 2007

机译：使用果蝇果蝇数据来发现人类中与疾病相关的蛋白质相互作用
5. Analysis of the downstream promoter element in Drosophila melanogaster and humans. [D] . Boulay, Thomas M. 2005

机译：果蝇和人类下游启动子元件的分析。
6. Human TTBK1 TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau [O] . Josefin Fernius, Annika Starkenberg, Malgorzata Pokrzywa, 2017

机译：人Tau蛋白的共表达加剧了果蝇中人TTBK1TTBK2和MARK1激酶的毒性
7. Human TTBK1, TTBK2 and MARK1 kinase toxicity in Drosophila melanogaster is exacerbated by co-expression of human Tau [O] . Fernius, Josefin, Starkenberg, Annika, Pokrzywa, Malgorzata, 2017

机译：人Tau蛋白的共表达加剧了果蝇中人TTBK1，TTBK2和MARK1激酶的毒性

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