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首页> 外文期刊>Chinese Medical Journal >Influence of bladder outlet obstruction and detrusor contractility on residual urine in patients with benign prostatic hyperplasia
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Influence of bladder outlet obstruction and detrusor contractility on residual urine in patients with benign prostatic hyperplasia

机译:前列腺增生患者膀胱出口梗阻和逼尿肌收缩力对残余尿的影响

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摘要

Objective To study the relationship between the degree of bladder outlet obstruction (BOO), detrusor contractility and residual urine in patients suffering from benign prostatic hyperplasia (BPH). Methods In 181 patients with BPH, degree of BOO, detrusor contractility, residual urine caculated from cathetering combined with the difference between the filling and the voiding were recorded and analysized statistically using urodynamic technique. Results Residual urine increased when the detusor contractility was weakened (F = 12.134, P = 0.001). In patients wih severe BOO, there was no significant difference in residual urine (F = 2.386, P = 0.071). Conclusions Increased residual urine is mainly resulted from decreased detrusor contractility. BOO has no significant influence on residual urine. Some patients with normal or weakened detrusor contractility may have more residual urine.
机译:目的探讨良性前列腺增生(BPH)患者膀胱出口梗阻程度(BOO),逼尿肌收缩力与残余尿量之间的关系。方法对181例BPH患者的BOO程度,逼尿肌收缩力,导管积尿,充盈与排尿的差异进行记录,并用尿流动力学技术进行统计分析。结果当氘化肌收缩力减弱时,残余尿量增加(F = 12.134,P = 0.001)。严重BOO的患者,残余尿量无显着差异(F = 2.386,P = 0.071)。结论残余尿量增加主要是由于逼尿肌收缩力降低。 BOO对残留尿液没有显着影响。逼尿肌收缩力正常或减弱的一些患者可能残留尿量更多。

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