机译:钌甲基咪唑配合物通过线粒体内在途径诱导肺癌A549细胞凋亡
Department of Chemistry, Jinan University, Guangzhou 510632, China;
School of Pharmacy, Guangdong Medical College, Zhanjiang 524023, China;
Department of Chemistry, Jinan University, Guangzhou 510632, China;
Department of Chemistry, Jinan University, Guangzhou 510632, China;
Department of Chemistry, Jinan University, Guangzhou 510632, China;
Department of Chemistry, Jinan University, Guangzhou 510632, China;
Department of Chemistry, Jinan University, Guangzhou 510632, China;
Institute of Life and Health Engineering, Jinan University, Guangzhou 510632, China;
ruthenium; apoptosis; caspase; Bcl-2; mitochondria;
机译:So-Cheong-Ryong-Tang通过激活非小细胞肺癌A549细胞的内在和外在凋亡通路以及抑制PI3K / Akt信号通路来诱导凋亡
机译:Calotropis Gigantea提取物通过在A549和NCI-H1299非小细胞肺癌细胞中产生的外部/内在途径和反应性氧物种产生凋亡
机译:H9通过非小细胞肺癌A549细胞的内在途径诱导凋亡。
机译:花生白蛋白诱导肺癌A549细胞凋亡的机制
机译:蛋氨酸应激诱导Cdc6和复制前复合体的下调并激活乳腺癌细胞的固有凋亡。
机译:硒-β-乳球蛋白通过内在的线粒体途径诱导人肺癌A549细胞凋亡
机译:Selenious-β-乳酰叶蛋白通过内在的线粒体途径诱导人肺癌A549细胞的凋亡