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机译:羧基末端调节蛋白的重复气溶胶传递可抑制K-ras〜(LA1)小鼠肺部肿瘤。
Laboratory of Toxicology, College of Veterinary Medicine, Seoul National University, Seoul, Korea;
Laboratory of Toxicology, College of Veterinary Medicine, Seoul National University, Seoul, Korea Nano Systems Institute-National Core Research Center, Seoul National University, Seoul, Korea;
Laboratory of Toxicology, College of Veterinary Medicine, Seoul National University, Seoul, Korea;
Laboratory of Toxicology, College of Veterinary Medicine, Seoul National University, Seoul, Korea;
Department of Chemistry,College of Natural Science, Seoul National University, Seoul, Korea;
Department of Chemistry,College of Natural Science, Seoul National University, Seoul, Korea;
Division of Endocrinology, Metabolism and Lipids, Emory University School of Medicine, Atlanta, Georgia;
Laboratory of Toxicology, College of Veterinary Medicine, Seoul National University, Seoul, Korea Nano Systems Institute-National Core Research Center, Seoul National University, Seoul, Korea;
lentivirus; carboxyl-terminal modulator protein; lung cancer; K-ras~(LA1) mice; aerosol gene delivery;
机译:慢病毒介导的O-糖基化突变体骨桥蛋白的气溶胶递送抑制了K-ras(LA1)小鼠的肺肿瘤发生。
机译:慢病毒介导的K-ras无效小鼠肺中的气溶胶介导的羧基末端调节蛋白基因转染。
机译:shAkt1和Pdcd4的双重表达抑制K-ras(LA1)小鼠的肺肿瘤发生。
机译:生物相容性SPE-alt-PEG / Akt1 shRNA复合物在肺癌基因治疗的K-ras〜(LA1)肺癌模型小鼠中的气溶胶递送
机译:K-ras基因多态性不是小鼠肺肿瘤易感性差异的关键
机译:羧基末端调节蛋白的重复气溶胶传递抑制K-rasLA1小鼠肺部肿瘤。
机译:慢病毒介导的羧基 - 末端调节剂蛋白基因转染通过气溶胶在K-Ras零小鼠的肺中