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机译:紫外线致癌过程中小鼠角质形成细胞中Fas-配体表达的丧失
From the Department of Immunology,The University of Texas M. D. Anderson Cancer Center, Houston, Texas;
From the Department of Immunology,The University of Texas M. D. Anderson Cancer Center, Houston, Texas;
and the Department of Pathology,University of Tasmania, Hobart, Tasmania, Australia;
From the Department of Immunology,The University of Texas M. D. Anderson Cancer Center, Houston, Texas;
From the Department of Immunology,The University of Texas M. D. Anderson Cancer Center, Houston, Texas;
From the Department of Immunology,The University of Texas M. D. Anderson Cancer Center, Houston, Texas;
机译:小鼠角质形成细胞中Smad4的缺失导致对紫外线致癌性的敏感性增加,而ercc1介导的DNA修复减少
机译:角质形成细胞p38缺失抑制Ras诱导的肿瘤形成,而全身性p38缺失在小鼠皮肤化学致癌作用的早期增强皮肤炎症
机译:小鼠角质形成细胞中蛋白质磷酸酶6的丢失增加了对紫外线B致癌作用的敏感性
机译:非热血浆对人角质形成细胞的治疗可阻断TNF-α和IFN-γ介导的促炎基因表达
机译:CDK6和细胞周期蛋白D3在角质形成细胞增殖和小鼠皮肤癌变中的作用。
机译:紫外线致癌过程中小鼠角质形成细胞中Fas-配体表达的丧失
机译:紫外线致癌过程中小鼠角质形成细胞中Fas-配体表达的丧失