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机译:基质金属蛋白酶9启动子活性与肿瘤进展过程中的入侵诱导。
From the Departments of Pathology and Laboratory Medicine,University of Pennsylvania, Philadelphia, Pennsylvania|and Otorhinolaryngology,University of Pennsylvania, Philadelphia, Pennsylvania;
the Vision Research Laboratories,New England Eye Center, Tufts University School of Medicine, Boston, Massachusetts;
and the Departments of Biology and Pathology,Institute for Molecular Biology and Biotechnology, McMaster University, Hamilton, Ontario, Canada;
and Otorhinolaryngology,University of Pennsylvania, Philadelphia, Pennsylvania;
From the Departments of Pathology and Laboratory Medicine,University of Pennsylvania, Philadelphia, Pennsylvania;
From the Departments of Pathology and Laboratory Medicine,University of Pennsylvania, Philadelphia, Pennsylvania;
机译:肿瘤和基质金属蛋白酶对肿瘤进展,侵袭和转移的贡献。
机译:Mangiferin通过抑制核因子-κB活性抑制肿瘤坏死因子-α-诱导的基质金属蛋白酶-9表达和细胞侵袭
机译:孕酮诱导的阻断因子通过改变基质金属蛋白酶活性来差异调节滋养细胞和肿瘤侵袭
机译:多光谱光声层析成像技术在非缺血性脑缺血小鼠模型中检测基质金属蛋白酶活性
机译:细胞外基质金属蛋白酶诱导剂(EMMPRIN)调节人和狒狒子宫内膜的基质金属蛋白酶
机译:基质金属蛋白酶9启动子活性与肿瘤进展过程中的入侵诱导。
机译:诱导基质金属蛋白酶9启动子活性与肿瘤进展期间的侵袭同时发生。