机译:磷酸化的p38作为TNF {α}诱导大肠肿瘤细胞凋亡的新型DAPK相互作用伴侣的鉴定。
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
and Molecular and Clinical Immunology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
and Molecular and Clinical Immunology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
and Molecular and Clinical Immunology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
and Molecular and Clinical Immunology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
and the Laboratory for Experimental Dermatology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
From the Institutes of Pathology,Department of Dermatology and Venerology, Otto-von-Guericke University Magdeburg, Magdeburg, Germany;
机译:在结直肠肿瘤细胞TNFα诱导的细胞凋亡中鉴定磷酸化P38作为新型DAPK - 相互作用伴侣(Vol 175,PG 557,2009)
机译:在大肠肿瘤细胞中由TNFα诱导的凋亡过程中磷酸化的p38作为新型DAPK相互作用伴侣的鉴定(第175卷,第557页,2009年)
机译:在TNFα诱导的结直肠肿瘤细胞凋亡过程中,磷酸化的p38被鉴定为新型的DAPK相互作用伴侣。
机译:TRAIL(与TNF相关的凋亡诱导配体)在酸性细胞外pH下诱导肿瘤细胞中的坏死状细胞死亡
机译:Rac1在TNF-α诱导的IEC-6细胞凋亡中的作用。
机译:在TNFα诱导的大肠肿瘤细胞凋亡过程中磷酸化的p38作为新型DAPK相互作用伙伴的鉴定。
机译:在TNFα诱导的大肠肿瘤细胞凋亡过程中磷酸化的p38作为新型DAPK相互作用伙伴的鉴定。