机译:小细胞骨架相关分子,成纤维细胞生长因子受体1癌基因伴侣2 /创伤诱导转录本3.0(FGFR1OP2 / wit3.0),促进成纤维细胞驱动的伤口闭合
From the Weintraub Center for Reconstructive Biotechnology,School of Dentistry, University of California, Los Angeles, Los Angeles, California|Division of Advanced Prosthodontics, Biomaterials and Hospital Dentistry, School of Dentistry, the Biomedical Engineering Interdepartmental Program,School of Dentistry, University of California, Los Angeles, Los Angeles, California;
From the Weintraub Center for Reconstructive Biotechnology,School of Dentistry, University of California, Los Angeles, Los Angeles, California;
From the Weintraub Center for Reconstructive Biotechnology,School of Dentistry, University of California, Los Angeles, Los Angeles, California|Henry Samueli School of Engineering and Applied Science, and the Section of Periodontics,School of Dentistry, University of California, Los Angeles, Los Angeles, California;
From the Weintraub Center for Reconstructive Biotechnology,School of Dentistry, University of California, Los Angeles, Los Angeles, California;
机译:细胞骨架相关的小分子,成纤维细胞生长因子受体1癌基因伴侣2 /伤口诱导转录本3.0(FGFR1OP2 / wit3.0),促进了成纤维细胞驱动的伤口闭合。
机译:小细胞骨架相关分子,成纤维细胞生长因子受体1癌基因伴侣2 /创伤诱导转录本3.0(FGFR1OP2 / wit3.0),促进成纤维细胞驱动的伤口闭合
机译:准备闭合伤口的辅助手段:高压氧,生长因子,皮肤替代品,负压伤口治疗(真空辅助闭合)。
机译:碱性成纤维细胞生长因子的深入研究促进皮肤伤口修复的有效机制
机译:损伤诱导的P2Y受体刺激导致表皮生长因子受体激活和角膜上皮伤口愈合
机译:小细胞骨架相关分子成纤维细胞生长因子受体1癌基因伴侣2 /创伤诱导转录本3.0(FGFR1OP2 / wit3.0)促进成纤维细胞驱动的伤口闭合
机译:小细胞骨架相关分子,成纤维细胞生长因子受体1癌基因伴侣2 /创伤诱导转录本3.0(FGFR1OP2 / wit3.0),促进成纤维细胞驱动的伤口闭合