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Numerical Simulation of Asymmetrically Altered Growth as Initiation Mechanism of Scoliosis

机译:非对称生长变化作为脊柱侧弯发生机制的数值模拟

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摘要

The causes of idiopathic scoliosis are still uncertain; buckling is mentioned often, but never proven. The authors hypothesize another option: unilateral postponement of growth of MM Rotatores or of ligamentum flavum and intertransverse ligament. In this paper, both buckling and the two new theories of scoliotic initiation are studied using a new finite element model that simulates the mechanical behavior of the human spine. This model was validated by the stiffness data of Panjabi et al. (J. Biomech. 9:185–192, 1976). After a small correction of the prestrain of some ligaments and the MM Rotatores the model appeared to be valid. The postponement in growth was translated in the numerical model in an asymmetrical stiffness. The spine was loaded axially and the resulting deformation was analyzed for the presence of the coupling of lateral deviation and axial rotation that is characteristic for scoliosis. Only unilateral postponement of growth of ligamentum flavum and intertransverse ligament appeared to initiate scoliosis. Buckling did not initiate scoliosis.
机译:特发性脊柱侧弯的病因尚不确定。屈曲经常被提及,但从未得到证实。作者假设了另一种选择:单侧推迟MM旋转肌或黄韧带和横断间韧带的生长。在本文中,使用模拟人类脊柱力学行为的新型有限元模型研究了屈曲和脊柱侧弯起始的两种新理论。该模型已通过Panjabi等人的刚度数据验证。 (J. Biomech。9:185–192,1976)。在对一些韧带和MM Rotatores的预应力进行少量校正之后,该模型似乎是有效的。增长的延迟在数值模型中以不对称的刚度转化。对脊柱进行轴向加载,并分析由此产生的变形,以了解是否存在侧弯和轴向旋转耦合(这是脊柱侧弯的特征)。仅单方面推迟黄韧带和横向横韧带的生长才开始脊柱侧弯。屈曲并未引发脊柱侧弯。

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