首页> 美国卫生研究院文献>The Journal of Biological Chemistry >Withdrawal: myo-Inositol oxygenase overexpression accentuates generation of reactive oxygen species and exacerbates cellular injury following high glucose ambience: A new mechanism relevant to the pathogenesis of diabetic nephropathy.

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Withdrawal: myo-Inositol oxygenase overexpression accentuates generation of reactive oxygen species and exacerbates cellular injury following high glucose ambience: A new mechanism relevant to the pathogenesis of diabetic nephropathy.

机译：戒断：肌醇加氧酶的过度表达加剧了活性氧的产生并在高葡萄糖气氛后加剧了细胞损伤：与糖尿病性肾病发病机理相关的新机制。

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期刊名称 The Journal of Biological Chemistry
作者
Lin Sun; Rajesh K. Dutta; Ping Xie; Yashpal S. Kanwar;
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作者单位

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年(卷),期 2019(294),26
年度 2019
页码 10380
总页数 1
原文格式 PDF
正文语种
中图分类分子生物学;
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1. Increased expression of myo-inositol oxygenase is involved in the tubulointerstitial injury of diabetic nephropathy. [J] . Lu Y, Liu C, Miao X Experimental and clinical endocrinology and diabetes: Official journal, German Society of Endocrinology [and] German Diabetes Association . 2009,第6期

机译：肌醇加氧酶表达增加与糖尿病肾病的肾小管间质损伤有关。
2. Modulation of renal-specific oxidoreductase/myo-inositol oxygenase by high-glucose ambience. [J] . Nayak B, Xie P, Akagi S, Proceedings of the National Academy of Sciences of the United States of America . 2005,第50期

机译：高糖环境对肾脏特异性氧化还原酶/肌醇氧化酶的调节。
3. Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy. [J] . Susztak K, Raff AC, Schiffer M, Diabetes . 2006,第1期

机译：葡萄糖诱发的活性氧在糖尿病性肾病发作时引起足细胞凋亡和足细胞耗竭。
4. Mitochondrial Injury Caused by Reactive Oxygen Species Generation under High Fluence Low-Power Laser Irradiation Treatment [C] . Shengnan Wu, Lei Huang, Xuegang Sun, Biophotonics and immune responses IV . 2009

机译：高通量低功率激光辐照下活性氧产生引起线粒体损伤
5. Cellular mechanisms of the systemic inflammatory response following resuscitated hemorrhagic shock: The role of reactive oxygen species and Toll-like receptor 4. [D] . Powers, Kinga Antonina. 2008

机译：失血性休克复苏后全身炎症反应的细胞机制：活性氧和Toll样受体的作用4。
6. myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience [O] . Lin Sun, Rajesh K. Dutta, Ping Xie, 2016

机译：肌醇加氧酶过表达加速了活性氧的产生并加剧了高糖环境后的细胞损伤
7. myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience [O] . Lin Sun, Rajesh K. Dutta, Ping Xie, 2016

机译：肌醇氧气氧酶过表达突出了活性氧物种的产生，高葡萄糖氛围后加剧了细胞损伤

Withdrawal: myo-Inositol oxygenase overexpression accentuates generation of reactive oxygen species and exacerbates cellular injury following high glucose ambience: A new mechanism relevant to the pathogenesis of diabetic nephropathy.

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