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Embolization of Two Occluder Devices Following Device Closure of Acute Postoperative Mitral Paravalvular Leak

机译:在急性术后二尖瓣瓣膜静脉泄漏的装置闭合后两种封闭装置的栓塞

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摘要

A 36-year-old male underwent mitral valve replacement with a single leaflet mechanical prosthesis for rheumatic chronic severe mitral regurgitation associated with cardiac cachexia, chronic atrial fibrillation, and severe pulmonary hypertension. His postoperative period was complicated by uncontrolled heart failure, recurrent refractory pericardial and pleural effusions, warranting creation of surgical pleuro pericardial window through a left anterolateral thoracotomy. A mild mitral paravalvular leak adjacent to the left atrial appendage on third post-operative day progressively enlarged to 16 mm at 2 months (Figure 1, Movie 1) leading to referral to our centre. Cardiac catheterization after 2 months showed near systemic pulmonary artery pressures. The left atrial v-waves and mean pressures after transseptal puncture were 86 and 46 millimetres of mercury (Figure 2). Closure of the leak through transseptal sheaths (Figure 3, Movie 2) with 2 large 16 mm and 10 mm Amplatzer muscular ventricular septal defect occluders (Abbott, Plymouth, MN, USA) reduced the left atrial pressures to 35 millimetres of mercury. However, both devices embolized within a few minutes and started floating like butterflies in the left atrium due to the impact of the paravalvular regurgitation jet (Figure 4, Movie 3). Immediate surgical retrieval on cardiopulmonary bypass through a redo sternotomy and suture of the leak led to early recovery. Risk factors for embolization of paravalvular leak devices include very large regurgitant orifices, more than one interlocking device and inadequate annular fibrosis in an acute postoperative setting.1),2) On a follow-up of 6 years, he was asymptomatic with normal prosthesis function and permanent atrial fibrillation.
机译:一名36岁的男性接受二尖瓣替代,具有单一宣传叶机械假体,具有与心脏恶病毒,慢性心房颤动和严重肺动脉高压有关的风湿慢性重症二尖瓣反流性。他的术后期通过不受控制的心力衰竭,复发性耐火心包和胸腔积液复杂化,通过左前任胸廓切开术治疗手术胸膜外皮窗口。在第三次术后日左侧心房附近的轻度二分高瓣膜泄漏逐渐扩大到16毫米,在2个月(图1,电影1),导致我们的中心推荐。 2个月后心脏导管显示出在全身性肺动脉压力附近。旋转穿刺后的左心房V波和平均压力为86和46毫米的汞(图2)。通过旋转鞘盖关闭泄漏(图3,电影2),具有2个大16毫米和10 mm放大器肌肉室间隔缺陷封堵器(Abbott,Plymouth,Mn,USA)将左心室压低降低到35毫米的汞。然而,两种装置都在几分钟内栓塞,并且由于静脉瓣膜瓣膜射流的影响(图4,电影3)的影响,左侧庭的蝴蝶开始漂浮。通过重做胸骨切开术和泄漏的缝合导致早期恢复,立即手术检索。瓣膜泄漏装置的抗栓塞风险因素包括非常大的反斜孔,令人互动的术后设定中的一个互锁装置和不足的环形纤维化。2)在6年的随访中,他与正常假体功能无症状和永久性心房颤动。

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