首页> 美国卫生研究院文献>Human Molecular Genetics >Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models
【2h】

Smcr8 deficiency disrupts axonal transport-dependent lysosomal function and promotes axonal swellings and gain of toxicity in C9ALS/FTD mouse models

机译:在C9ALS / FTD小鼠模型中Smcr8缺乏症破坏了依赖轴突运输的溶酶体功能并促进了轴突肿胀和毒性增加

代理获取
本网站仅为用户提供外文OA文献查询和代理获取服务,本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文,但由于OA文献来源多样且变更频繁,仍可能出现获取不到、文献不完整或与标题不符等情况,如果获取不到我们将提供退款服务。请知悉。

摘要

Since article was originally published online, some minor corrections which had been missed at the proof stage (due to OUP error) have since been updated: 1) In Fig, 5F, WT, and Smcr8-/- have been added into the labels, and also *, n.s. in the notes; 2) In Fig. 9C, GR has been amended to GP following a re-calculation, because the authors found that previous GR antibody recognizes non-specific autofluorescent proteins. The modifications did not change the conclusions and interpretation of the results.
机译:自从文章最初在线发布以来,由于在更新阶段由于OUP错误而错过了一些小更正:1)在图5F,WT和Smcr8-/-中已添加到标签中,还有*,ns在笔记; 2)在图9C中,由于作者发现先前的GR抗体识别非特异性自体荧光蛋白,因此在重新计算后将GR修改为GP。修改并没有改变结论和结果解释。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
代理获取

客服邮箱:kefu@zhangqiaokeyan.com

京公网安备:11010802029741号 ICP备案号:京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有
  • 客服微信

  • 服务号