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Enteropathogenic Escherichia coli (EPEC) Recruitment of PAR Polarity Protein Atypical PKCζ to Pedestals and Cell–Cell Contacts Precedes Disruption of Tight Junctions in Intestinal Epithelial Cells

机译：肠致病性大肠埃希菌（EPEC）招募对基座和细胞间接触的PAR极性蛋白非典型PKCζ导致肠上皮细胞中的紧密连接破坏

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期刊名称 International Journal of Molecular Sciences
作者
Rocio Tapia; Sarah E. Kralicek; Gail A. Hecht;
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作者单位

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年(卷),期 2020(21),2
年度 2020
页码 -1
总页数 20
原文格式 PDF
正文语种
中图分类分子生物学;
关键词
enteropathogenic;

机译：肠致病性;

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专利

1. Lactobacillus plantarum surface layer adhesive protein protects intestinal epithelial cells against tight junction injury induced by enteropathogenic Escherichia coli [J] . Zhihua Liu, Tongyi Shen, Peng Zhang, Molecular Biology Reports . 2011,第5期

机译：植物乳杆菌表面层粘附蛋白可保护肠上皮细胞免受由肠致病性大肠杆菌引起的紧密连接损伤
2. Lactobacillus plantarum surface layer adhesive protein protects intestinal epithelial cells against tight junction injury induced by enteropathogenic Escherichia coli [J] . Liu Zhihua, Shen Tongyi, Zhang Peng, Molecular biology reports . 2011,第5期

机译：植物乳杆菌表面层粘附蛋白可保护肠上皮细胞免受肠道致病性大肠杆菌诱导的紧密连接损伤
3. Enteropathogenic Escherichia coli infection leads to appearance of aberrant tight junctions strands in the lateral membrane of intestinal epithelial cells [J] . Muza-Moons MM, Schneeberger EE, Hecht GA Cellular microbiology . 2004,第8期

机译：肠致病性大肠埃希菌感染导致肠上皮细胞外侧膜出现紧密连接的异常链
4. CAPSAICIN-ENHANCED RIBOSOMAL PROTEIN P2 EXPRESSION AND RECOVERY OF TIGHT JUNCTION PERMEABILITY IN HUMAN INTESTINAL Caco-2 CELLS [C] . Junkyu Han, Mitsuaki Akutsu, Terence P. N. Talorete, Annual Meeting of the Japanese Association for Animal Cell Technology . 2006

机译：辣椒素增强的核糖体蛋白P2在人肠道Caco-2细胞中的紧密结渗透性的表达和恢复
5. Mechanisms of intestinal epithelial tight junction disruption by enteropathogenic Escherichia coli. [D] . Muza-Moons, Michelle Marie. 2007

机译：肠致病性大肠杆菌肠上皮紧密连接破坏的机制。
6. Tricellular Tight Junction Protein Tricellulin Is Targeted by the Enteropathogenic Escherichia coli Effector EspG1 Leading to Epithelial Barrier Disruption [O] . Vijay Morampudi, Franziska A. Graef, Martin Stahl, 2017

机译：三细胞紧密连接蛋白Tricellulin被肠致病性大肠杆菌效应EspG1靶向导致上皮屏障破坏。
7. Outer membrane vesicles and soluble factors released by probiotic Escherichia coli Nissle 1917 and commensal ECOR63 enhance barrier function by regulating expression of tight junction proteins in intestinal epithelial cells [O] . Carina Shianya Alvarez, Josefa Badia, Manel Bosch, 2016

机译：益生菌大肠杆菌Nissle 1917和共生ECOR63释放的外膜囊泡和可溶性因子通过调节肠上皮细胞中紧密连接蛋白的表达来增强屏障功能

Enteropathogenic Escherichia coli (EPEC) Recruitment of PAR Polarity Protein Atypical PKCζ to Pedestals and Cell–Cell Contacts Precedes Disruption of Tight Junctions in Intestinal Epithelial Cells

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