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A growing chain of evidence linking genetic variation in angiotensinogen with essential hypertension: focus on A haplotype of human angiotensinogen gene containing −217A increases blood pressure in transgenic mice compared with −217G by Jain et al.

机译：越来越多的证据将血管紧张素原的遗传变异与原发性高血压联系起来：专注于含有-217A的人类血管紧张素原基因的单倍型与-217G相比增加了转基因小鼠的血压Jain等人。

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期刊名称 American Journal of Physiology - Regulatory Integrative and Comparative Physiology
作者
Curt D. Sigmund;
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作者单位

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年(卷),期 -1(295),6
年度 -1
页码 R1846–R1848
总页数 10
原文格式 PDF
正文语种
中图分类人体生理学 ;
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1. A growing chain of evidence linking genetic variation in angiotensinogen with essential hypertension: focus on "a haplotype of human angiotensinogen gene containing -217A increases blood pressure in transgenic mice compared with -217G," by Jain et al. [J] . Curt D. Sigmund American Journal of Physiology . 2008 ,第6aPta2期

机译：越来越多的证据将血管紧张素原的遗传变异与原发性高血压联系起来：Jain等人关注“与-217G相比，含有-217A的人类血管紧张素原基因的单倍型增加了转基因小鼠的血压”。
2. A growing chain of evidence linking genetic variation in angiotensinogen with essential hypertension: focus on "A haplotype of human angiotensinogen gene containing -217A increases blood pressure in t [J] . Curt D. Sigmund American Journal of Physiology . 2009 ,第6aPta2期

机译：越来越多的证据将血管紧张素原的遗传变异与原发性高血压联系起来：着眼于“含有-217A的人类血管紧张素原基因单倍型会增加血压
3. A growing chain of evidence linking genetic variation in angiotensinogen with essential hypertension: focus on "A haplotype of human angiotensinogen gene containing -217A increases blood pressure in t [J] . Curt D. Sigmund American Journal of Physiology . 2008 ,第6aPta2期

机译：一种越来越多的证据链，将血管紧张素引的遗传变异与必需的高血压：关注“含有-217A的人血管紧张素基因的单倍型增加了T中的血压
4. A haplotype of human angiotensinogen gene containing −217A increases blood pressure in transgenic mice compared with −217G [O] . Sudhir Jain, Govindaiah Vinukonda, Steven N. Fiering, -1

机译：与-217G相比包含-217A的人类血管紧张素原基因单倍型可增加转基因小鼠的血压
5. A haplotype of human angiotensinogen gene containing −217A increases blood pressure in transgenic mice compared with −217G [O] . Jain, Sudhir, Vinukonda, Govindaiah, Fiering, Steven N., 2008

机译：与-217G相比，包含-217A的人类血管紧张素原基因单倍型可增加转基因小鼠的血压
6. Exercise, genetics, and blood pressure: Focus on 'Physical Exercise and Blood Pressure With Reference to the Angiotensinogen M285T Polymorphism' and on 'Angiotensinogen M235T Polymorphism Associates With Exercise Hemodynamics in Postmenopausal Women' [R] . Sonna, L. A. , Glueck, S. B. , Jeunemaitre, X. 2002

机译：运动，遗传和血压：关注“参与血管紧张素原m285T多态性的体育锻炼和血压”以及“血管紧张素原m235T多态性与绝经后妇女运动血流动力学的关联”

A growing chain of evidence linking genetic variation in angiotensinogen with essential hypertension: focus on A haplotype of human angiotensinogen gene containing −217A increases blood pressure in transgenic mice compared with −217G by Jain et al.

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