首页> 美国卫生研究院文献>American Journal of Physiology - Cell Physiology >Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells

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Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells

机译：血管紧张素II 1型受体通过多种机制调节RhoA和Rho激酶/ ROCK活化。专注于血管紧张素II通过依赖SHP2的血管平滑肌细胞中RhoGAP p190A的去磷酸化诱导RhoA激活

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期刊名称 American Journal of Physiology - Cell Physiology
作者
Keita Kimura; Satoru Eguchi;
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作者单位

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年(卷),期 -1(297),5
年度 -1
页码 C1059–C1061
总页数 6
原文格式 PDF
正文语种
中图分类细胞生物学;人体生理学;
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1. Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on “Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells” [J] . Keita Kimura, Satoru Eguchi American Journal of Physiology . 2009,第5aPta1期

机译：血管紧张素II型受体通过多种机制调节RHOA和RHO-激酶/岩石活化。专注于“血管紧张素II通过在血管平滑肌细胞中的rhogap p190a的SHP2依赖性去磷酸化诱导RHOA活化”
2. Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells. [J] . Bregeon J, Loirand G, Pacaud P American Journal of Physiology . 2009,第5aPta1期

机译：血管紧张素II通过血管平滑肌细胞中的rhogap p190a的SHP2依赖性去磷酸化诱导RHOA活化。
3. Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells. [J] . Bregeon J, Loirand G, Pacaud P American Journal of Physiology . 2009,第5aPta1期

机译：血管紧张素II通过SHP2依赖性RhoGAP p190A在血管平滑肌细胞中的SHP2依赖性去磷酸化诱导RhoA活化。
4. Nitric Oxide-Angiotensin II Interaction in Modulation of Vascular Smooth Muscle Signalling: A Possible Role for ROS? [C] . Siobhan Kelleher, Stephen Wilson, Alan K. Keenan NATO Advanced Study Institute on Vascular Endothelium : Mechanisms of Cell Signaling . 1999

机译：一氧化氮 - 血管紧张素II在调节血管平滑肌信号中的相互作用：ROS的可能作用？
5. Novel mechanisms of cardioprotection: Catecholamine release by natriuretic peptides: Characterization and prevention; activation of ALDH2 in mast cell mitochondria: Prevention of local angiotensin II formation; activation of histamine H3-receptors: Reduction of angiotensin AT1-receptor neuronal expression [D] . Chan, Noel Yan-ki 2012

机译：心脏保护的新机制：利钠肽释放儿茶酚胺：表征和预防；肥大细胞线粒体中ALDH2的激活：预防局部血管紧张素II的形成； H3受体的活化：降低血管紧张素AT1受体神经元表达
6. Adiponectin Attenuates Angiotensin II-Induced Vascular Smooth Muscle Cell Remodeling through Nitric Oxide and the RhoA/ROCK Pathway [O] . Wared Nour-Eldine, Crystal M. Ghantous, Kazem Zibara, 2016

机译：脂联素通过一氧化氮和RhoA / ROCK途径减弱血管紧张素II诱导的血管平滑肌细胞重塑。
7. Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on “Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells” [O] . Kimura, Keita, Eguchi, Satoru 2009

机译：血管紧张素II 1型受体通过多种机制调节RhoA和Rho激酶/ ROCK活化。专注于“血管紧张素II通过依赖SHP2的血管平滑肌细胞中RhoGAP p190A的去磷酸化诱导RhoA激活”

Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells

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