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The acceleration of glucose accumulation in renal cell carcinoma assessed by FDG PET/CT demonstrated acquisition of resistance to tyrosine kinase inhibitor therapy

机译：通过FDG PET / CT评估的肾细胞癌中葡萄糖积累的加速表明获得了对酪氨酸激酶抑制剂治疗的耐药性

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BackgroundTyrosine-kinase inhibitor (TKI) targeting angiogenesis improves the prognosis of patients with metastatic renal cell carcinoma (RCC), but its effect is temporary. In order to understand the mechanism by which RCC acquires resistance to TKI, we investigated the change of glucose accumulation in RCC by FDG PET/CT when they demonstrated progression disease (PD) against TKI.

机译：背景靶向血管生成的酪氨酸激酶抑制剂（TKI）可改善转移性肾细胞癌（RCC）患者的预后，但其作用是暂时的。为了了解RCC抵抗TKI的机制，我们研究了当FDG PET / CT表现出针对TKI的疾病（PD）时，RCD中葡萄糖积累的变化。

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期刊名称 BMC Cancer
作者
Noboru Nakaigawa; Keiichi Kondo; Daiki Ueno; Kazuhiro Namura; Kazuhide Makiyama; Kazuki Kobayashi; Koichi Shioi; Ichiro Ikeda; Takeshi Kishida; Tomohiro Kaneta; Ryogo Minamimoto; Ukihide Tateishi; Tomio Inoue; Masahiro Yao;
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年(卷),期 2017(17),-1
年度 2017
页码 39
总页数 9
原文格式 PDF
正文语种
中图分类肿瘤学;
关键词
Renal cell carcinoma Tyrosine kinase inhibitor Resistance acquisition FDG PET/CT Standardized uptake value;

机译：肾细胞癌;酪氨酸激酶抑制剂;耐药性获得;FDG PET / CT;标准化摄取值;

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专利

1. The acceleration of glucose accumulation in renal cell carcinoma assessed by FDG PET/CT demonstrated acquisition of resistance to tyrosine kinase inhibitor therapy [J] . Noboru Nakaigawa, Keiichi Kondo, Daiki Ueno, BMC Cancer . 2017,第1期

机译：通过FDG PET / CT评估的肾细胞癌中葡萄糖积累的加速表明获得了对酪氨酸激酶抑制剂治疗的耐药性
2. FDG PET/CT as imaging biomarker in the era of molecular targeting therapies: sequential FDG PET/CT demonstrated biological response and acquisition of resistance to tyrosine kinase inhibitor therapy for renal cell carcinoma [J] . Nakaigawa N., Namura K., Ueno D., European journal of cancer: official journal for European Organization for Research and Treatment of Cancer (EORTC) [and] European Association for Cancer Research (EACR) . 2014,第Suppla6期

机译：FDG PET / CT作为分子靶向治疗时代的成像生物标志物：连续的FDG PET / CT证明了肾细胞癌的生物学应答和对酪氨酸激酶抑制剂治疗的耐药性
3. The early response of renal cell carcinoma to tyrosine kinase inhibitors evaluated by FDG PET/CT was not influenced by metastatic organ [J] . Manabu Kakizoe, Masahiro Yao, Ukihide Tateishi, BMC Cancer . 2014,第1期

机译：FDG PET / CT评估肾细胞癌对酪氨酸激酶抑制剂的早期反应不受转移器官的影响
4. The Epidermal Growth Factor Receptor Tyrosine Kinase Inhibitor ZD1839 (Iressa) Suppresses Proliferation and Invasion of Human Oral Squamous Carcinoma Cells via p53 Independent and MMP, uPAR Dependent Mechanism [C] . EUN JU LEE, JIN HA WHANG, NAM KYEONG JEON, Meeting on Cell Signaling World: Signal Transduction Pathways as Therapeutic Targets . 2007

机译：表皮生长因子受体酪氨酸激酶抑制剂ZD1839（Iressa）通过独立于p53和MMP，uPAR依赖性机制抑制人口腔鳞状细胞癌细胞的增殖和侵袭
5. The effect of photodynamic therapy (PDT) on epidermal growth factor receptor and interleukin-6 cytokine signaling, and tyrosine kinase inhibitors enhance the efficacy of PDT by increasing intracellular accumulation of photosensitizers. [D] . Liu, Weiguo. 2005

机译：光动力疗法（PDT）对表皮生长因子受体和白介素6细胞因子信号转导的影响，酪氨酸激酶抑制剂通过增加光敏剂在细胞内的积累来增强PDT的功效。
6. Early assessment by FDG-PET/CT of patients with advanced renal cell carcinoma treated with tyrosine kinase inhibitors is predictive of disease course [O] . Daiki Ueno, Masahiro Yao, Ukihide Tateishi, 2012

机译：FDG-PET / CT对酪氨酸激酶抑制剂治疗的晚期肾细胞癌的早期评估可预示疾病进程
7. The acceleration of glucose accumulation in renal cell carcinoma assessed by FDG PET/CT demonstrated acquisition of resistance to tyrosine kinase inhibitor therapy [O] . 2017

机译：通过FDG PET / CT评估的肾细胞癌中葡萄糖积累的加速表明获得了对酪氨酸激酶抑制剂治疗的耐药性

The acceleration of glucose accumulation in renal cell carcinoma assessed by FDG PET/CT demonstrated acquisition of resistance to tyrosine kinase inhibitor therapy

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