首页> 美国卫生研究院文献>Clinical and Experimental Immunology >DC‐SIGN reacts with TLR‐4 and regulates inflammatory cytokine expression via NF‐κB activation in renal tubular epithelial cells during acute renal injury

【2h】

DC‐SIGN reacts with TLR‐4 and regulates inflammatory cytokine expression via NF‐κB activation in renal tubular epithelial cells during acute renal injury

机译：在急性肾损伤期间DC-SIGN与TLR-4发生反应并通过NF-κB激活调节肾小管上皮细胞中炎性细胞因子的表达

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

获取外文期刊封面目录资料

页面导航

著录项
引文网络
相似文献
相关主题

著录项

期刊名称 Clinical and Experimental Immunology
作者
D. Feng; Y. Wang; Y. Liu; L. Wu; X. Li; Y. Chen; Y. Chen; Y. Chen; C. Xu; K. Yang; T. Zhou;
展开▼
作者单位

展开▼
年(卷),期 2018(191),1
年度 2018
页码 107–115
总页数 9
原文格式 PDF
正文语种
中图分类免疫学;
关键词
acute kidney injury DC‐SIGN NF‐κB renal tubular epithelial cells TLR‐4;

机译：急性肾损伤;DC-SIGN;NF-κB;肾小管上皮细胞;TLR-4;

相似文献

外文文献
中文文献
专利

1. DC‐SIGN reacts with TLR‐4 and regulates inflammatory cytokine expression via NF‐κB activation in renal tubular epithelial cells during acute renal injury [J] . Feng D., Wang Y., Liu Y., Clinical and Experimental Immunology: An Official Journal of the British Society for Immunology . 2018,第1期

机译：DC-符号用TLR-4反应，并在急性肾损伤期间通过NF-κB活化调节肾脏管状上皮细胞中的炎性细胞因子表达
2. Lentivirus-Mediated Silencing of Src Homology 2 Domain-Containing Protein Tyrosine Phosphatase 2 Inhibits Release of Inflammatory Cytokines and Apoptosis in Renal Tubular Epithelial Cells Via Inhibition of the TLR4/NF-kB Pathway in Renal Ischemia-Reperfusion Injury [J] . Teng J.-F., Wang K., Jia Z.-M., Kidney and blood pressure research . 2018,第4期

机译：慢病毒介导的Src同源性2域蛋白酪氨酸磷酸酶2沉默通过抑制TLR4 / NF-kB途径抑制肾缺血再灌注损伤炎性细胞因子的释放和肾小管上皮细胞的凋亡。
3. CD200R1 agonist attenuates LPS-induced inflammatory response in human renal proximal tubular epithelial cells by regulating TLR4-MyD88-TAK1-mediated NF-kappa B and MAPK pathway [J] . Ding Yan, Yang Huilan, Xiang Wei, Biochemical and Biophysical Research Communications . 2015,第2期

机译：CD200R1激动剂通过调节TLR4-MyD88-Tak1介导的NF-Kappa B和Mapk途径，衰减人肾近端管状上皮细胞中的LPS诱导的炎症反应
4. THE EXPRESSION OF COX-2 AND PRO-INFLAMMATORY CYTOKINES INDUCED BY PCB IN HUMAN MAST CELLS REQUIRES NF-k kk kB ACTIVATION [C] . O-Kyun Kwon, Eunkyung Lee, Tae Chul Moon, International symposium on halogenated environmental organic pollutants and persistent organic pollutants and POPs;DIOXIN;DIOXIN 2001;POPs . 2001

机译：PCB诱导的乳腺癌细胞中COX-2的表达和促炎性细胞因子的激活需要NF-k kk kB的激活
5. The JAK2/Y343/STAT 5 signaling axis is required for erythropoietin-mediated protection against ischemic injury in renal tubular epithelial cells. [D] . Breggia, Anne C. 2008

机译：JAK2 / Y343 / STAT 5信号轴是促红细胞生成素介导的抗肾小管上皮细胞缺血性损伤的保护所必需的。
6. Knockdown of ANGPTL2 Protects Renal Tubular Epithelial Cells Against Hypoxia/Reoxygenation-Induced Injury via Suppressing TLR4/NF-κB Signaling Pathway and Activating Nrf2/HO-1 Signaling Pathway [O] . Heli Xiang, Wujun Xue, Yang Li, 2020

机译：Angptl2的敲低通过抑制TLR4 / NF-κB信号传导途径和激活NRF2 / HO-1信号通路保护肾小管上皮细胞免受缺氧/雷诺诱导的损伤
7. DC-SIGN reacts with TLR-4 and regulates inflammatory cytokine expression via NF-κB activation in renal tubular epithelial cells during acute renal injury [O] . D. Feng, Y. Wang, Y. Liu, 2017

机译：DC-符号用TLR-4反应，并在急性肾损伤期间通过NF-κB活化调节肾脏管状上皮细胞中的炎性细胞因子表达

DC‐SIGN reacts with TLR‐4 and regulates inflammatory cytokine expression via NF‐κB activation in renal tubular epithelial cells during acute renal injury

著录项

引文网络

相似文献

相关主题

期刊订阅