首页> 美国卫生研究院文献>Journal of Virology >Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens

【2h】

Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens

机译：在感染过程中出现的高度致病性猿猴免疫缺陷病毒变种会增强感染力并能够下调CD4而不是I类主要组织相容性复合抗原

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

著录项
相似文献
相关主题

著录项

期刊名称 Journal of Virology
作者
Parul G. Patel; Monica T. Yu Kimata; Julia E. Biggins; Joelle M. Wilson; Jason T. Kimata;
展开▼
作者单位

展开▼
年(卷),期 2002(76),17
年度 2002
页码 8977
总页数 2
原文格式 PDF
正文语种
中图分类人体病毒学（致病病毒）;病毒（滤过性病毒）;
关键词
入库时间 2022-08-17 11:45:36

相似文献

外文文献
专利

1. Analysis of Gag-specific Cytotoxic T Lymphocytes in Simian Immunodeficiency Virus–infected Rhesus Monkeys by Cell Staining with a Tetrameric Major Histocompatibility Complex Class I–Peptide Complex [J] . Marcelo J. Kuroda, J?rn E. Schmitz, Dan H. Barouch, The Journal of Experomental Medicine . 1998,第9期

机译：用四聚体主要组织相容性复合物I类-肽复合物进行细胞染色，分析猿猴免疫缺陷病毒感染的恒河猴中的Gag特异性细胞毒性T淋巴细胞
2. Analysis of Gag-specific Cytotoxic T Lymphocytes in Simian Immunodeficiency Virus–infected Rhesus Monkeys by Cell Staining with a Tetrameric Major Histocompatibility Complex Class I–Peptide Complex [J] . Marcelo J. Kuroda, J?rn E. Schmitz, Dan H. Barouch, The Journal of Experomental Medicine . 1998,第9期

机译：用四聚体主要组织相容性复合物I类-肽复合物进行细胞染色，分析猿猴免疫缺陷病毒感染的恒河猴中的Gag特异性细胞毒性T淋巴细胞
3. Major histocompatibility complex class I-restricted cytotoxic T lymphocyte responses during primary simian immunodeficiency virus infection in Burmese rhesus macaques. [J] . Nakamura M, Takahara Y, Ishii H, Microbiology and Immunology . 2011,第11期

机译：主要的组织相容性复杂I类限制了在猕猴猕猴的原发猿猴免疫缺陷病毒感染过程中的细胞毒性T淋巴细胞反应。
4. Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens [O] . Parul G. Patel, Monica T. Yu Kimata, Julia E. Biggins, 2002

机译：在感染过程中出现的高度致病性猿猴免疫缺陷病毒变种会增强感染力并能够下调CD4而不是I类主要组织相容性复合抗原
5. Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens [O] . Patel, Parul G., Yu Kimata, Monica T., Biggins, Julia E., 2002

机译：在感染过程中出现的高度致病性猿猴免疫缺陷病毒变种会增强感染力，并能够下调CD4而不是I类主要组织相容性复合抗原

Highly Pathogenic Simian Immunodeficiency Virus mne Variants That Emerge during the Course of Infection Evolve Enhanced Infectivity and the Ability To Downregulate CD4 but Not Class I Major Histocompatibility Complex Antigens

著录项

相似文献

相关主题

期刊订阅