猕猴桃果实腐烂病的发病规律及药剂筛选试验

摘要

Experiments were conducted on infection regularity of fruit rot on Kiwifruit by Phomopsis sp.with fruit bagging and on toxicity of eight fungicides against the pathogen was determined by mycelial growth rate in laboratory.The results showed that infection started about three weeks after blossom fall and the heaviest infection was about six weeks after blossom fall.The experiments on fungi toxicity indicated that EC50 of hexaconazole ME and prochloraz-manganese chloride complex WP reached 0.068 and 0.105 μg/mL,indicating the highest inhibitive effect against Phomopsis sp.EC50 of difenoconazole WG,thiophanate-Methyl WP,and diniconazole WP,were 0.224,0.307,0.426 μg/mL.EC50 of Mancozeb WP,chlorothalonil WP,and triadimefon EC were 7.862,36.65 and 99.04 μg/mL,showing less control effects.%采用套袋方法研究了猕猴桃果实腐烂病病菌拟茎点霉(Phomopsis sp.)的侵染规律,采用菌丝生长速率法测定了甲基硫菌灵可湿性粉剂等8种杀菌剂的毒力,结果表明,该菌在谢花后的3周左右开始侵染幼果,6周左右侵染达到高峰;己唑醇微乳剂、咪酰胺锰盐可湿性粉剂对猕猴桃果实腐烂病菌菌丝具有较高的抑制活性,其EC50 分别为0.068、0.105 μg/mL,其次是苯醚甲环唑水分散剂、甲基硫菌灵可湿性粉剂和烯唑醇可湿性粉剂,其EC50 分别为0.224、0.307和0.426 μg/mL,代森锰锌可湿性粉剂、百菌清可湿性粉剂、三唑酮乳油抑制活性较低,其EC50分别为7.862、36.65和99.04μg/ml.