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建筑节能用EPS与XPS

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摘要

建筑节能保温隔热用膨胀聚苯乙烯泡沫塑料板简称为聚苯板，分为模塑板(EPS)与挤塑板(XPS)两种。本文阐述了建筑节能保温用聚苯乙烯泡沫塑料板EPS与XPS的异同点。

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《2008年中国国际墙体保温材料及应用技术交流会》|2008年|48-53|共6页
会议地点杭州
作者

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作者单位

中国建筑学会;

中国建筑业协会;

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会议组织
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原文格式 PDF
中图分类 TU111.41;TU551.2;
关键词
建筑节能; 墙体保温; 保温材料; 聚苯乙烯;

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1. 建筑节能用EPS与XPS分析 [J] . 章银祥 . 墙材革新与建筑节能 . 2008 ,第005期
2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011 ,第4期
3. Long-term omeprazole and esomeprazole treatment does not significantly increase gastric epithelial cell proliferation and epithelial growth factor receptor expression and has no effect on apoptosis and p53 expression [J] . Istvan Hritz ,Laszlo Herszenyi ,Bela Molnar . 世界胃肠病学杂志：英文版 . 2005 ,第30期
4. Experimental study on therapeutic effect of in vivo expression of Cell Ⅰ-Hep Ⅱ recombinant polypeptide of fibronectin on murine H22 hepatocellular carcinoma [J] . Gui-Mei Zhang Yan Yang Bo Huang Hui Xiao Dong Li Zuo-Hua Feng Department of Biochemistry and Molecular BiologyTongji Medical CollegeHuazhong University of Science and TechnologyWuhan 430030Hubei ProvinceChina . 世界胃肠病学杂志：英文版 . 2003 ,第9期
5. 从运行数据分析绿色建筑节能途径——以江苏省建筑节能和绿色建筑示范区示范项目为例 [C] . Li Xianglin ,李湘 . 第十二届国际绿色建筑与建筑节能大会 . 2016
6. 影响居住建筑节能设计因素的分析与研究——青岛居住建筑节能设计初探 [A] . 牛帅 . 2013

建筑节能用EPS与XPS

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