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首页> 外文期刊>Journal of clinical neuroscience: official journal of the Neurosurgical Society of Australasia >Posterior decompression salvages Bryan total disc arthroplasty in post-operatively recurrent uncoforaminal stenosis
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Posterior decompression salvages Bryan total disc arthroplasty in post-operatively recurrent uncoforaminal stenosis

机译:后减压挽救Bryan全椎间盘置换术在术后复发性无孔眼狭窄中的应用

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摘要

We report on 69 retrospectively reviewed patients who received 73 Bryan (Medtronic Sofamor Danek, Memphis, TN, USA) total disc arthroplasties for recent soft cervical disc herniations over a 9.3 year period. Three patients returned with radiculopathy due to the redevelopment of uncoforaminal stenosis at the Bryan segment and later underwent posterior decompression of the uncoforaminal area without modification to the prosthesis. They recovered from the radiculopathy after decompression; however, one patient later required adjacent segment fusion to recover from concomitant cervicalgia. After posterior decompression, all prostheses continued to function normally. In one patient, however, bony bridging of the prosthesis is imminent, despite being currently asymptomatic. We normally exclude patients with uncoforaminal stenosis from Bryan arthroplasty. Analysis of three of these patients (4.3% of patients, 4.1% of prostheses) revealed that they received a prosthesis despite slight uncoforaminal stenosis (slight stenosis was known prior to surgery in one instance, two others were only discovered intra-operatively). Our observation raises the suspicion that slight uncoforaminal stenosis could also recur in physiologically working arthroplasty segments, and that in some instances this spur formation may progress into prosthesis bridging. However, more research is required to confirm the significance of uncoforaminal stenosis discovered pre- or intra-operatively in arthroplasty patients. Posterior minimally invasive decompression using the Frykholm-Scoville keyhole approach successfully treats uncoforaminal stenosis without revising the prosthesis.
机译:我们报告了69例经回顾性检查的患者,这些患者在9.3年的时间里接受了73例Bryan(Medtronic Sofamor Danek,美国田纳西州孟菲斯)的全椎间盘置换术。三名患者因布莱恩节段的非椎间孔狭窄再发展而再次发生神经根病,随后对非椎间孔区域进行了后路减压,但未对假体进行任何改动。减压后他们从神经根病中康复了。然而,一名患者后来需要邻近节段融合以从伴随的颈痛中恢复。后路减压后,所有假体继续正常运行。然而,在一名患者中,尽管目前无症状,但假体的骨桥迫在眉睫。我们通常将不伴小孔狭窄的患者从布莱恩关节置换术中排除。对其中三名患者的分析(4.3%的患者,4.1%的假体)显示,尽管有轻微的非椎间孔狭窄,但他们仍接受了假体(在手术前已知轻微狭窄,在手术中仅发现了另外两个)。我们的观察结果令人怀疑,轻微的非椎间孔狭窄也可能在生理上可行的关节置换术部分中复发,并且在某些情况下,这种骨刺形成可能会发展成假体桥接。但是,需要更多的研究来证实在关节置换术患者术前或术中发现的非椎间孔狭窄的重要性。使用Frykholm-Scoville锁孔入路的后路微创减压术无需矫正假体即可成功治疗非椎间孔狭窄。

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