Downregulation of SEMA4C Inhibit Epithelial-Mesenchymal Transition (EMT) and the Invasion and Metastasis of Cervical Cancer Cells via Inhibiting Transforming Growth Factor-beta 1 (TGF-β1)-Induced Hela cells p38 Mitogen-Activated Protein Kinase (MAPK) Activation

机译：下调SEMA4C通过抑制转化生长因子-β1（TGF-β1）诱导的Hela细胞p38丝裂原激活的蛋白激酶（MAPK）激活抑制上皮-间质转化（EMT）和宫颈癌细胞的侵袭和转移

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期刊名称 Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
作者
Lilan Yang; Yayuan Yu; Zhenfang Xiong; Hongxia Chen; Buzhen Tan; Hui Hu;
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作者单位

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年(卷),期 2020(26),-1
年度 2020
页码 -1
总页数 11
原文格式 PDF
正文语种
中图分类
关键词
MAP Kinase Signaling System; Neoplasm Invasiveness; Neoplasm Metastasis; Semaphorins; Transforming Growth Factor beta; Uterine Cervical Neoplasms;

机译：MAP激酶信号传导系统;肿瘤浸润;肿瘤转移;信号灯;转化生长因子β;子宫宫颈肿瘤;

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专利

1. Transforming growth factor-beta 1 (TGF-beta)-induced apoptosis of prostate cancer cells involves Smad7-dependent activation of p38 by TGF-beta-activated kinase 1 and mitogen-activated protein kinase kinase 3 [J] . Edlund S., Bu SH., Schuster N., Molecular biology of the cell . 2003,第2期

机译：转化生长因子-beta 1（TGF-beta）诱导的前列腺癌细胞凋亡涉及TGF-beta激活的激酶1和有丝分裂原激活的蛋白激酶3的Smad7依赖性激活p38。
2. Flavopiridol suppresses tumor necrosis factor-induced activation of activator protein-1, c-Jun N-terminal kinase, p38 mitogen-activated protein kinase (MAPK), p44/p42 MAPK, and Akt, inhibits expression of antiapoptotic gene products, and enhances apoptosis through cytochrome c release and caspase activation in human myeloid cells (Retraction of vol 73, pg 1549, 2008) [J] . Takada Y., Sethi G., Sung B., Molecular pharmacology. . 2016,第1期

机译：黄酮哌啶醇抑制肿瘤坏死因子诱导的活化蛋白1，c-Jun N端激酶，p38丝裂原活化蛋白激酶（MAPK），p44 / p42 MAPK和Akt的激活，抑制抗凋亡基因产物的表达并增强凋亡通过人类髓样细胞中细胞色素c的释放和胱天蛋白酶的活化（Retract of vol 73，pg 1549，2008）
3. Genistein inhibits matrix metalloproteinase type 2 activation and prostate cancer cell invasion by blocking the transforming growth factor beta-mediated activation of mitogen-activated protein kinase-activated protein kinase 2-27-kDa heat shock prote [J] . Xu L, Bergan RC Molecular pharmacology. . 2006,第3期

机译：金雀异黄素通过阻断转化生长因子β介导的丝裂原活化蛋白激酶活化蛋白激酶2-27-kDa热休克蛋白的表达，抑制基质金属蛋白酶2型激活和前列腺癌细胞的侵袭。
4. Modeling Of p38 Mitogen-activated Protein Kinase Inhibitors Using The CatalystTM HypoGen And k-nearest Neighbor QSAR Methods [C] . . 2005

机译：使用CatalystTM HypoGen和k近邻QSAR方法对p38丝裂原活化的蛋白激酶抑制剂进行建模
5. Identifying Novel Inhibitors of Epithelial to Mesenchymal Transition (EMT) for Targeting Pancreatic Cancer Metastasis and Cancer Stem Cells [D] . Polireddy, Kishore. 2015

机译：确定靶向胰腺癌转移和癌症干细胞的新型上皮间质转化抑制剂（EMT）
6. Transforming Growth Factor-β1 (TGF-β)–induced Apoptosis of Prostate Cancer Cells Involves Smad7-dependent Activation of p38 by TGF-β-activated Kinase 1 and Mitogen-activated Protein Kinase Kinase 3 [O] . Sofia Edlund, Shizhong Bu, Norbert Schuster, 2003

机译：转化生长因子-β1（TGF-β）诱导的前列腺癌细胞凋亡涉及TGF-β激活的激酶1和丝裂原激活的蛋白激酶3的Smad7依赖性激活p38。
7. Downregulation of SEMA4C Inhibit Epithelial-Mesenchymal Transition (EMT) and the Invasion and Metastasis of Cervical Cancer Cells via Inhibiting Transforming Growth Factor-beta 1 (TGF-β1)-Induced Hela cells p38 Mitogen-Activated Protein Kinase (MAPK) Activation [O] . Lilan Yang, Yayuan Yu, Zhenfang Xiong, 2020

机译：通过抑制转化生长因子-β1（TGF-β1）抑制SEMA4C的下调抑制上皮 - 间充质转换（EMT）和宫颈癌细胞的侵袭和转移 - 诱导的HELA细胞P38丝裂剂活化蛋白激酶（MAPK）活化

Downregulation of SEMA4C Inhibit Epithelial-Mesenchymal Transition (EMT) and the Invasion and Metastasis of Cervical Cancer Cells via Inhibiting Transforming Growth Factor-beta 1 (TGF-β1)-Induced Hela cells p38 Mitogen-Activated Protein Kinase (MAPK) Activation

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