process for separating the restlaengen of the verkaufslaenge divided walzstaeben

• 1. 一种斯泰尔摩控冷工艺方法 [P] . 中国专利： CN101664764B . 2011.08.31
• 2. 一种斯泰尔摩控冷工艺方法 [P] . 中国专利： CN101664764A . 2010-03-10
• 3. process for separating the restlaengen of the verkaufslaenge divided walzstaeben [P] . 法国专利： FR2201140B1 . 1978-01-06

  机译： 分离沃尔斯施泰本的剩余能量的方法

• 4. process for separating the restlaengen of the verkaufslaenge divided walzstaeben [P] . 德国专利： DE000002248177A . 1974-04-11

  机译： 分离沃尔斯施泰本的剩余能量的方法

• 5. process for separating the restlaengen of the verkaufslaenge divided walzstaeben [P] . 日本专利： JPS4971577A . 1974-07-10

  机译： 分离沃尔斯施泰本的剩余能量的方法

• 1. Simulated annealing-based optimal design of energy efficient ternary extractive dividing wall distillation process for separating benzene-isopropanol-water mixtures [J] . Min Li, Yue Cui, Xiaojing Shi, 中国化学工程学报（英文版） . 2021,第005期
• 2. Dividing Process in Base-10 Number System Reversed Dividing Process for 1/X [J] . Nick Huang 数学和系统科学：英文版 . 2015,第007期
• 3. Simulation Optimization and Experimental Study of Cross-Wall Adiabatic Dividing Wall Column Used to Separate Hexane-Heptane-Octane System [J] . Hu Yuqi, Fang Jing, Li Chunli 中国炼油与石油化工（英文版） . 2015,第001期
• 4. Optimal Structure of Vapor Recompression Heat Pump Dividing-wall Column for Separating Narrow Boiling Point Mixture [C] . Xiang Yiding, Huang Kejin Youth Academic Annual Conference of Chinese Association of Automation . 2021
• 5. An Online Algorithm for Separating Sparse and Low-Dimensional Signal Sequences From Their Sum, and Its Applications in Video Processing [D] . Guo, Han. 2019
• 6. Distinct Constrictive Processes Separated in Time and Space Divide Caulobacter Inner and Outer Membranes [O] . Ellen M. Judd, Luis R. Comolli, Joseph C. Chen, 2005
• 7. Distinct Constrictive Processes, Separated in Time and Space, Divide Caulobacter Inner and Outer Membranes† [O] . Judd, Ellen M., Comolli, Luis R., Chen, Joseph C., 2005

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. A corpus-based comparison of the use of divide and separate [J] . Ma Zhiyan . 快乐阅读(上旬刊) . 2011,第001期
• 3. 沃尔德斯连续流砂过滤器在某污水处理厂升级改造项目中运行报告分析 [C] . 北京沃尔德斯水务科技有限公司 . 2017中国污水处理厂提标改造高级研讨会 . 2017
• 4. 非线性沃尔泰拉延迟积分微分方程几类数值方法的散逸性 [A] . 姚金然 . 2007