Converse striking construction method null of underground body

• 1. 一种可折叠共轴反桨无人机及打击方法 [P] . 中国专利： CN109405649B . 2021.03.26
• 2. 多飞行器反拦截协同打击的制导方法、装置和飞行器 [P] . 中国专利： CN112782984A . 2021-05-11
• 3. Construction construction method null of the underground structure due to converse striking [P] . 日本专利： JP2555837B2 . 1996-11-20

  机译： 反击对地下结构的施工施工方法无效

• 4. Using the long span elevator, construction construction method null of the underground body body due to the converse hitting construction method which conveys the materials [P] . 日本专利： JP2767481B2 . 1998-06-18

  机译： 使用大跨度电梯，由于传送物料的逆向撞击施工方法，地下车身的施工方法无效

• 5. Inputting construction method null of the underground floor stand rising concrete due to converse striking [P] . 日本专利： JP2797967B2 . 1998-09-17

  机译： 反击法输入地下楼架抬高混凝土的施工方法无效

• 1. Surrounding rock deformation analysis of underground caverns with multi-body finite element method [J] . Wan-jin LIANG, Chao SU, Fei WANG, 水科学与水工程：英文版 . 2009,第003期
• 2. Diagnosing Graves’ Disease and Non-Graves Hyperthyroidism Using TSH Receptor Antibody Test versus Non-TSH Receptor Antibody Test Methods of Diagnosis [J] . Mohammed Qader Meena 内分泌与新陈代谢疾病期刊（英文） . 2020,第002期
• 3. CONVERSE ANALYSIS OF THE EXPANSION FORCES OF MALL ROCKS OF UNDERGROUND CONSTRUCTION [C] . Xu Qingda World congress on non-metallic minerals;WCNMM 1989 . 1989
• 4. Fast solution of large-body problems using domain decomposition and null-field generation in the method of moments. [D] . Killian, Tyler N. 2009
• 5. Burns caused by striking underground electricity cables. [O] . M J Timmons 1973
• 6. On the optimization of the construction of ventilation systems during the construction of metro by underground method [O] . Alexander Vartanov, Irina Markova, Anatoly Fedash 2019
• 7. Review of Underground Construction Methods and Opening Stability for Repositories in Clay/Shale Media, Fuel Cycle Research and Development [R] . Hardin, EL 2014

• 1. Surrounding rock deformation analysis of underground caverns with multi-body finite element method [J] . Wan-jin LIANG ,Chao SU ,Fei WANG . 水科学与水工程 . 2009,第003期
• 2. Introduction of the Direct Method by Illustrating Schrdinger Equation and Its Application to Wave-Wave Striking Interaction [J] . 高守亭 ,刘坤儒 . 大气科学进展：英文版 . 1990,第2期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 不同压力作用下体外反搏对剪切应力频率分量的影响 [C] . 王怀阳 ,戴丽 ,郑振声 . 2006首届国际体外反搏学术交流会 . 2006
• 5. 《打击伊斯兰极端分子恐怖主义的非军事战略：以往反暴恐行动中的经验教训》翻译的项目报告 [A] . 刘晓童 . 2015