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Stochastic Resonance Controlled Upregulation of Internal Noise after Hearing Loss as a Putative Cause of Tinnitus-Related Neuronal Hyperactivity

机译：随机共振控制听力损失后内部噪声上调是耳鸣相关神经元过度活动的一个推测因素

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Patrick Krauss; Konstantin Tziridis; Claus Metzner; Achim Schilling; Ulrich Hoppe; Holger Schulze;
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1. Development of tinnitus-related neuronal hyperactivity through homeostatic plasticity after hearing loss: a computational model. [J] . Schaette R, Kempter R The European Journal of Neuroscience . 2006,第11期

机译：听力损失后通过稳态可塑性发展与耳鸣有关的神经元活动亢进：一种计算模型。
2. Noise overexposure alters long-term somatosensory-auditory processing in the dorsal cochlear nucleus-possible basis for tinnitus-related hyperactivity? [J] . DehmelS., PradhanS., KoehlerS., The Journal of Neuroscience: The Official Journal of the Society for Neuroscience . 2012,第5期

机译：噪声过度暴露会改变耳蜗背核中的长期体感听觉处理吗？可能是耳鸣相关活动亢进的基础吗？
3. EEG rhythms lateralization patterns in children with unilateral hearing loss are different from the patterns of normal hearing controls during speech-in-noise listening [J] . Cartocci Giulia, Scorpecci Alessandro, Borghini Gianluca, Hearing Research: An International Journal . 2019,第期

机译：单方面听力损失的儿童的EEG节奏横向化模式与语音噪声监听期间的正常听力控制模式不同
4. Stochastic Resonance in Excitable Neuronal System with Phase-Noise [C] . Xiaoming Liang, Liang Zhao Advances in brain inspired cognitive systems. . 2012

机译：具有相位噪声的兴奋性神经元系统中的随机共振
5. Modification of the Hearing In Noise Test (HINT) for evaluating amplification in listeners with high-frequency hearing loss. [D] . Dybala, Paul David. 2004

机译：修改了听觉噪声测试（HINT），以评估具有高频听力损失的听众的放大率。
6. Stochastic Resonance Controlled Upregulation of Internal Noise after Hearing Loss as a Putative Cause of Tinnitus-Related Neuronal Hyperactivity [O] . Patrick Krauss, Konstantin Tziridis, Claus Metzner, 2016

机译：耳聋相关神经元活动亢进的可能原因是听力损失后随机共振控制的内部噪声上调。
7. Development of tinnitus-related neuronal hyperactivity through homeostatic plasticity after hearing loss: a computational model [O] . Richard Kempter 2014

机译：听力损失后通过稳态可塑性发展耳鸣相关神经元过度活跃：计算模型

Stochastic Resonance Controlled Upregulation of Internal Noise after Hearing Loss as a Putative Cause of Tinnitus-Related Neuronal Hyperactivity

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