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Aberrant Type I Interferon Regulation in Autoimmunity: Opposite Directions in MS and SLE, Shaped by Evolution and Body Ecology

机译：自身免疫中异常的I型干扰素调节：ms和sLE的相反方向，由进化和身体生态学决定

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Anthony T. Reder; Xuan Feng;
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1. BIIB059, a monoclonal antibody targeting BDCA2, decreases Type I Interferon-related genes transcriptional activity in subjects with systemic lupus erythematosus (SLE) and active cutaneous LE (CLE) [J] . Werth V. P., Merola J. F., Furie R., The Journal of investigative dermatology. . 2018,第5a19期

机译：靶向BDCA2的单克隆抗体BiiB059，降低了具有全身性狼疮（SLE）和活性皮肤le（CLE）的受试者的I型干扰素相关基因转录活性。
2. Animal Virus Ecology and Evolution Are Shaped by the Virus Host-Body Infiltration and Colonization Pattern [J] . Jan Slingenbergh Pathogens . 2019,第2期

机译：动物病毒的生态学和进化受病毒宿主渗透和定居模式的影响
3. The interplay of type I and type II interferons in murine autoimmune cholangitis as a basis for sex‐biased autoimmunity [J] . Bae Heekyong R., Hodge Deborah L., Yang Guo‐Xiang, Hepatology: Official Journal of the American Association for the Study of Liver Diseases . 2018,第4期

机译：鼠自身免疫性胆管炎I型和II型干扰素的相互作用作为性别偏见的自身免疫基础
4. LC-MS/MS-BASED CHARACTERIZATION OF GLIOBLASTOMA CELL RESPONSES TO TYPE I INTERFERONS FOR ESTABLISHING DEFECTS IN ANTIVIRAL MECHANISMS [C] . Irina Tarasova, Alesya Tereshkova, Elizaveta Solovyeva, International Mass Spectrometry Conference . 2018

机译：基于LC-MS / MS的胶质母细胞瘤细胞对I型干扰素的表征，用于在抗病毒机制中建立缺陷
5. Type I Interferons Negatively Regulate Foxp3+ Regulatory T Cells during Acute Viral Infection and Autoimmunity [D] . Srivastava, Shivani 2014

机译：I型干扰素在急性病毒感染和自身免疫过程中负调控Foxp3 +调节性T细胞。
6. Aberrant Type I Interferon Regulation in Autoimmunity: Opposite Directions in MS and SLE Shaped by Evolution and Body Ecology [O] . Anthony T. Reder, Xuan Feng 2013

机译：自身免疫中异常的I型干扰素调节：MS和SLE的相反方向受进化和人体生态学影响
7. Aberrant Type I Interferon Regulation in Autoimmunity: Opposite directions in MS and SLE, shaped by Evolution and Body Ecology [O] . Anthony T Reder, Xuan eFeng 2013

机译：自身免疫中异常的I型干扰素调节：ms和sLE的相反方向，由进化和身体生态学塑造

Aberrant Type I Interferon Regulation in Autoimmunity: Opposite Directions in MS and SLE, Shaped by Evolution and Body Ecology

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