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Norepinephrine-induced potentiation is not blocked by the local diffusion of the NMDA channel blocker, ketamine, at concentrations that prevent tetanically-induced long-term potentiation in the dentate gyrus in vivo

机译：去甲肾上腺素诱导的增强作用不会被NMDA通道阻滞剂氯胺酮的局部扩散所阻断，其浓度应能防止替丁酸引起的齿状回在体内的长期增强作用

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Thesis (M.Sc.)--Memorial University of Newfoundland, 1995. Psychology

机译：论文（硕士）-纽芬兰纪念大学，1995年。心理学

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Frizzell Lynn Marie 1967-;
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年度 1994
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正文语种 Eng
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1. Targeting of ribosomal protein S6 to dendritic spines by in vivo high frequency stimulation to induce long-term potentiation in the dentate gyrus Targeting of ribosomal protein S6 to dendritic spines by in vivo high frequency stimulation to induce long-term potentiation in the dentate gyrus Targeting of ribosomal protein S6 to dendritic spines by in vivo high frequency stimulation to induce long-term potentiation in the dentate gyrus [J] . Kaoru Inokuchi, Itsuko Nihonmatsu, Noriaki Ohkawa, Biology Open . 2015,第11期

机译：通过体内高频刺激将核糖体蛋白S6定向到树突棘，以诱导齿状回的长期增强。通过体内高频刺激将核糖体蛋白S6定向到树突棘，诱导在齿状回中的长期增强。核糖体蛋白S6通过体内高频刺激诱导齿状回长期增强
2. Long-term potentiation-like facilitation through GABAA receptor blockade in the mouse dentate gyrus in vivo. [J] . Matsuyama S, Taniguchi T, Kadoyama K, Neuroreport . 2008,第18期

机译：通过GABAA受体阻滞剂在小鼠齿状回体内的长期增强作用。
3. Developmental lead (Pb) exposure reduces the ability of the NMDA antagonist MK-801 to suppress long-term potentiation (LTP) in the rat dentate gyrus, in vivo. [J] . Gilbert ME, Lasley SM Neurotoxicology and teratology . 2007,第3期

机译：暴露于发育中的铅（Pb）会降低NMDA拮抗剂MK-801在体内抑制大鼠齿状回中的长期增强（LTP）的能力。
4. Experimental and modeling studies of the contribution of NMDA receptor-channels to the expression of long-term potentiation [C] . Wang Z., Song D., Berger T.W. Engineering in Medicine and Biology, 2002. 24th Annual Conference and the Annual Fall Meeting of the Biomedical Engineering Society EMBS/BMES Conference, 2002. Proceedings of the Second Joint . 2002

机译：NMDA受体通道对长期增强表达的贡献的实验和模型研究
5. Norepinephrine-induced potentiation is not blocked by the local diffusion of the NMDA channel blocker, ketamine, at concentrations that prevent tetanically-induced long-term potentiation in the dentate gyrus in vivo. [D] . Frizzell, Lynn Marie. 1994

机译：去甲肾上腺素诱导的增强作用不会被NMDA通道阻滞剂氯胺酮的局部扩散所阻断，其浓度可防止在体内齿状回中由强直性诱导的长期增强作用。
6. Sustained Arc/Arg3.1 Synthesis Controls Long-Term Potentiation Consolidation through Regulation of Local Actin Polymerization in the Dentate Gyrus In Vivo [O] . Elhoucine Messaoudi, Tambudzai Kanhema, Jonathan Soulé, 2007

机译：持续的Arc / Arg3.1合成通过调节齿状回体内的局部肌动蛋白聚合来控制长期增强。
7. N-methyl-d-aspartate receptor independent changes in expression of polysialic acid-neural cell adhesion molecule despite blockade of homosynaptic long-term potentiation and heterosynaptic long-term depression in the awake freely behaving rat dentate gyrus [O] . Rodríguez J. J., Dallérac G. M., Tabuchi M., 2009

机译：N-甲基-d-天冬氨酸受体独立改变聚唾液酸 - 神经细胞粘附分子的表达，尽管在清醒自由行为的大鼠齿状回中阻断同源突触长期增强和异突触长期抑制
8. Modulation of Long-Term Potentiation and Epileptiform Activity in the Rat Dentate Gyrus by the Group II Metabotropic Glutamate Receptor Subtype mGluR3 [R] . Lea, P. M. 2000

机译：II组代谢型谷氨酸受体亚型mGluR3对大鼠齿状回长期增强和癫痫样活动的调节

Norepinephrine-induced potentiation is not blocked by the local diffusion of the NMDA channel blocker, ketamine, at concentrations that prevent tetanically-induced long-term potentiation in the dentate gyrus in vivo

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