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Human macrophage inflammatory protein-3α/CCL20/LARC/Exodus/SCYA20 is transcriptionally upregulated by tumor necrosis factor-α via a non-standard NF-κB site

机译：人类巨噬细胞炎性蛋白3α/ CCL20 / LARC / Exodus / SCYA20通过非标准NF-κB位点被肿瘤坏死因子-α转录上调

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Harant Hanna; Eldershaw Suzy A.; Lindley Ivan J.D.;
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年度 2001
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1. Human macrophage inflammatory protein‐3α/CCL20/LARC/Exodus/SCYA20 is transcriptionally upregulated by tumor necrosis factor‐α via a non‐standard NF‐κB site [J] . Eldershaw Suzy A., Harant Hanna, Lindley Ivan J.D. FEBS Letters . 2001,第3期

机译：人类巨噬细胞炎性蛋白-3α/ CCL20 / LARC /出埃及记/ SCYA20通过非标准的NF-κB位点被肿瘤坏死因子-α转录上调
2. Synergistic Induction of Macrophage Inflammatory Protein-3α/CCL20 Production by Interleukin-17A and Tumor Necrosis Factor-α in Nasal Polyp Fibroblasts [J] . Manabu Nonaka, Nozomu Ogihara, Akira Fukumoto, World Allergy Organization Journal . 2009,第10期

机译：白细胞介素17A和肿瘤坏死因子-α在鼻息肉成纤维细胞中协同诱导巨噬细胞炎性蛋白3α/ CCL20的产生。
3. Genomic organization of the CC chemokine mip-3alpha/CCL20/larc/exodus/SCYA20, showing gene structure, splice variants, and chromosome localization. [J] . Nelson RT, Boyd J, Gladue RP, Genomics . 2001,第1期

机译：CC趋化因子mip-3alpha / CCL20 / larc / exodus / SCYA20的基因组组织，显示了基因结构，剪接变体和染色体定位。
4. Production of nitric oxide, tumor necrosis factor-α and interleukin-6 by RAW264.7 macrophage cells treated with lactic acid bacteria isolated from kimchi [C] . Haeng Jeon Hur, Ki Won Lee, Hyong Joo Lee International Conference on Food Factors . 2004

机译：用乳酸菌分离的乳酸菌治疗的乳酸细胞生产一氧化氮，肿瘤坏死因子-α和白细胞介素-6
5. Intracellular Signal Transduction Mechanisms Regulating the Activation of Human Bronchial Epithelial Cells by Interleukin-17A, Interleukin-27, Tumor Necrosis Factor-alpha and Human Basophils in Inflammatory Diseases [D] . Cao, Ju 2010

机译：调节人白细胞介素17A，白细胞介素27，肿瘤坏死因子-α和人类嗜碱性粒细胞在炎性疾病中激活人支气管上皮细胞的细胞内信号转导机制
6. Synergistic Induction of Macrophage Inflammatory Protein-3α/CCL20 Production by Interleukin-17A and Tumor Necrosis Factor-α in Nasal Polyp Fibroblasts [O] . Manabu Nonaka, Nozomu Ogihara, Akira Fukumoto, 2009

机译：白细胞介素17A和肿瘤坏死因子-α在鼻息肉成纤维细胞中协同诱导巨噬细胞炎性蛋白3α/ CCL20的产生。
7. Human T cell leukemia virus type-I Tax activates human macrophage inflammatory protein-3α/CCL20 gene transcription via the NF-κB pathway [O] . Yoshitaka Imaizumi, Shinichi Sugita, Kazuo Yamamoto, 2002

机译：人T细胞白血病病毒类型-I税通过NF-κB途径激活人巨噬细胞炎症蛋白-3α/ CCl20基因转录
8. Cytokine (Tumor Necrosis Factor, IL-6, and IL-8) Production by Respiratory Syncytial Virus-Infected Human Alveolar Macrophages [R] . Becker, S., Quay, J., Soukup, J. 1991

机译：呼吸道合胞病毒感染的人肺泡巨噬细胞产生细胞因子（肿瘤坏死因子，IL-6和IL-8）

Human macrophage inflammatory protein-3α/CCL20/LARC/Exodus/SCYA20 is transcriptionally upregulated by tumor necrosis factor-α via a non-standard NF-κB site

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