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PERK Activation Preserves the Viability and Function of Remyelinating Oligodendrocytes in Immune-Mediated Demyelinating Diseases

机译：PERK激活保留了免疫介导的脱髓鞘疾病中髓鞘少突胶质细胞的活力和功能。

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Lin Yifeng; Huang Guangcun; Jamison Stephanie; Li Jin; Harding Heather P.; Ron David; Lin Wensheng;
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1. PERK activation preserves the viability and function of remyelinating oligodendrocytes in immune-mediated demyelinating diseases [J] . LinY., HuangG., JamisonS., American Journal of Pathology: Official Publication of the American Association of Pathologists . 2014,第2期

机译：PERK激活保留了免疫介导的脱髓鞘疾病中髓鞘少突胶质细胞的活力和功能
2. PERK activation preserves the viability and function of remyelinating oligodendrocytes in immune-mediated demyelinating diseases [J] . Yifeng Lin, Guangcun Huang, Stephanie Jamison, The American journal of pathology. . 2014,第2期

机译：PERK激活保留了免疫介导的脱髓鞘疾病中髓鞘少突胶质细胞的活力和功能
3. IL-4-Induced Peroxisome Proliferator-Activated Receptor γ Activation Inhibits NF-κB Trans Activation in Central Nervous System (CNS) Glial Cells and Protects Oligodendrocyte Progenitors under Neuroinflammatory Disease Conditions: Implication for CNS-Demyelinating Diseases [J] . Ajaib S. Paintlia, Manjeet K. Paintlia, Inderjit Singh, The journal of immunology . 2006,第7期

机译：IL-4诱导的过氧化物酶体增殖物激活的受体γ激活抑制神经炎性疾病条件下中枢神经系统（CNS）胶质细胞中NF-κB反式激活并保护少突胶质祖细胞：对CNS脱髓鞘疾病的意义。
4. OLIGODENDROCYTE PROGENITOR CELLS: TARGETS TO IMPROVE CNS FUNCTION AND REPAIR IN AGEING AND DISEASE [C] . Abbe Crawford, Richa Tripathi, William Richardson, Conference of the American^College^of^Veterinary^Internal^Medicine . 2016

机译：oligodendrocyte祖细胞：改善CNS功能和衰老和疾病修复的靶标
5. Deriviation of homogeneous population of oligodendrocytes from embryonic stem cells: Therapeutic implications for white matter repair in demyelinating diseases and traumatic brain injury. [D] . Neman-Ebrahim, Yousha. 2009

机译：从胚胎干细胞获得少突胶质细胞的同质种群：脱髓鞘疾病和脑外伤中白质修复的治疗意义。
6. PERK Activation Preserves the Viability and Function of Remyelinating Oligodendrocytes in Immune-Mediated Demyelinating Diseases [O] . Yifeng Lin, Guangcun Huang, Stephanie Jamison, 2014

机译：PERK激活保留了免疫介导的脱髓鞘疾病中髓鞘少突胶质细胞的活力和功能。
7. Activating transcription factor 6α deficiency exacerbates oligodendrocyte death and myelin damage in immune-mediated demyelinating diseases [O] . Sarrabeth Stone, Shuangchan Wu, Stephanie Jamison, 2018

机译：激活转录因子6α缺乏加剧了免疫介导的脱髓鞘疾病中的少突胶鸡死亡和髓鞘损伤

PERK Activation Preserves the Viability and Function of Remyelinating Oligodendrocytes in Immune-Mediated Demyelinating Diseases

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