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Measurements of exocytosis from single presynaptic nerve terminals reveal heterogeneous inhibition by Ca2+-channel blockers

机译：测量单个突触前神经末梢的胞吐作用表明Ca2 +通道阻滞剂具有异质抑制作用

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Reuter Harald;
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1. Presynaptic Ca2+ influx and vesicle exocytosis at the mouse endbulb of Held: a comparison of two auditory nerve terminals. [J] . Lin KH, Oleskevich S, Taschenberger H The Journal of Physiology . 2011,第17期

机译：突触前Ca 2+流入和小鼠尾突处的囊泡胞吐作用：两个听觉神经末梢的比较。
2. Salvinorin A exerts opposite presynaptic controls on neurotransmitter exocytosis from mouse brain nerve terminals. [J] . Grilli M, Neri E, Zappettini S Neuropharmacology . 2009,第5a6期

机译：Salvinorin A对小鼠脑神经末梢的神经递质胞吐作用有相反的突触前控制。
3. Presynaptic mechanisms underlying the alpha-lipoic acid facilitation of glutamate exocytosis in rat cerebral cortex nerve terminals. [J] . Wang SJ, Chen HH Neurochemistry International: The International Journal for the Rapid Publication of Critical Reviews, Preliminary and Original Research Communications in Neurochemistry . 2007,第1期

机译：大鼠大脑皮质神经末梢谷氨酸胞吐作用促进α-硫辛酸的突触前机制。
4. Simulation of a Presynaptic Nerve Terminal with a Tethered Particle System Model [C] . Rhys Goldstein, Gabriel Wainer Annual International Conference of the IEEE Engineering in Medicine and Biology Society . 2009

机译：具有束缚粒子系统模型的突触前神经终端的模拟
5. Membrane capacitance measurements of depolarization-triggered exocytosis in single nerve terminals from the rat neurohypophysis [D] . Lim, Nancy Fidler 1991

机译：大鼠神经垂体单神经末梢去极化触发胞吐作用的膜电容测量
6. Presynaptic Capacitance Measurements and Ca2+ Uncaging Reveal Submillisecond Exocytosis Kinetics and Characterize the Ca2+ Sensitivity of Vesicle Pool Depletion at a Fast CNS Synapse [O] . Markus Wölfel, Ralf Schneggenburger 2003

机译：突触前电容测量和Ca2 +解笼揭示亚毫秒级胞吐动力学并表征快速CNS突触囊泡池耗竭的Ca2 +敏感性。
7. Presynaptic Ca2+ influx and vesicle exocytosis at the mouse endbulb of Held: A comparison of two auditory nerve terminals. [O] . Lin, K., Oleskevich, S., Taschenberger, H. 2011

机译：Held小鼠endbulb的突触前Ca2 +内流和囊泡胞吐：两个听神经末梢的比较。

Measurements of exocytosis from single presynaptic nerve terminals reveal heterogeneous inhibition by Ca2+-channel blockers

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