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ROS leads to MnSOD upregulation through ERK2 translocation and p53 activation in selenite-induced apoptosis of NB4 cells

机译：ROS通过亚硒酸盐诱导的NB4细胞凋亡通过ERK2易位和p53激活导致MnSOD上调

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Li Zhushi; Shi Kejian; Guan Liying; Cao Tingming; Jiang Qian; Yang Yang; Xu Caimin;
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年度 2010
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1. ROS leads to MnSOD upregulation through ERK2 translocation and p53 activation in selenite-induced apoptosis of NB4 cells. [J] . Li Z, Shi K, Guan L, FEBS letters. . 2010,第11期

机译：ROS通过亚硒酸盐诱导的NB4细胞凋亡通过ERK2易位和p53激活导致MnSOD上调。
2. ROS leads to MnSOD upregulation through ERK2 translocation and p53 activation in selenite‐induced apoptosis of NB4 cells [J] . FEBS Letters . 2010,第11期

机译：ROS通过亚硒酸盐诱导的NB4细胞凋亡通过ERK2易位和p53激活导致MnSOD上调
3. FV-429 Induced Apoptosis Through ROS-Mediated ERK2 Nuclear Translocation and p53 Activation in Gastric Cancer Cells [J] . Zhou Yuxin, Wei Libin, Zhang Haiwei, Journal of cellular biochemistry. . 2015,第8期

机译：FV-429通过ROS介导的ERK2核易位和p53激活在胃癌细胞中诱导凋亡。
4. Inhibitors of diverse metabolic steps cause increased apoptosis in deoxyadenosine-resistantmouse leukemia L1210 cells that lack p53 expression: convergence at caspase-3 activation [C] . Ann H. Cory, Caroline C. Edwards, Jennifer G. Hall, International Symposium on Regulation of Enzyme Activity and Synthesis in Normal and Neoplastic Tissues . 2003

机译：不同代谢步骤的抑制剂导致缺乏p53表达的脱氧腺苷抵抗疗效中的细胞凋亡增加：Caspase-3活化的收敛性
5. Stathmin Depletion from Cells Lacking p53 Leads to Delayed Cell Cycle Progression and Apoptosis Due to Increased Microtubule Stability. [D] . Carney, Bruce K. 2011

机译：缺乏p53的细胞中的stathmin耗竭会导致细胞周期进程的延迟和细胞凋亡，这归因于微管稳定性的提高。
6. Blocking NF-κB nuclear translocation leads to p53-related autophagy activation and cell apoptosis [O] . Bao-Song Zhu, Chun-Gen Xing, Fang Lin, 2011

机译：阻断NF-κB核易位导致p53相关的自噬激活和细胞凋亡
7. Blocking NF-κB nuclear translocation leads to p53-related autophagy activation and cell apoptosis [O] . Zhu, Bao-Song, Xing, Chun-Gen, Lin, Fang, 2011

机译：阻断NF-κB核易位导致p53相关的自噬激活和细胞凋亡

ROS leads to MnSOD upregulation through ERK2 translocation and p53 activation in selenite-induced apoptosis of NB4 cells

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