首页> 外文OA文献 >Romedepsin (RM), A Hdaci, Significantly Increases The Expression Of NKG2D Ligands, MIC A/B, In Leukemia/Lymphoma Cells (LL), In Part Through The Glycogen Synthase Kinase-3 (GSK-3) Pathway, Resulting In Enhanced NK Cytotoxicity: Translational Approach For Adoptive NK Cell Immunotherapy

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Romedepsin (RM), A Hdaci, Significantly Increases The Expression Of NKG2D Ligands, MIC A/B, In Leukemia/Lymphoma Cells (LL), In Part Through The Glycogen Synthase Kinase-3 (GSK-3) Pathway, Resulting In Enhanced NK Cytotoxicity: Translational Approach For Adoptive NK Cell Immunotherapy

机译：Romedepsin（RM），一种Hdaci，可显着增加白血病/淋巴瘤细胞（LL）中NKG2D配体MIC A / B的表达，部分通过糖原合酶激酶3（GSK-3）途径，导致增强的NK细胞毒性：过继性NK细胞免疫疗法的转化方法

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Satwani P.; Saha A.; Bavishi S.; Zhao F.; Ayello J.; van de Ven C.; Cairo M.S.;
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1. Inhibition of Glycogen Synthase Kinase-3 Increases NKG2D Ligand MICA Expression and Sensitivity to NK Cell–Mediated Cytotoxicity in Multiple Myeloma Cells: Role of STAT3 [J] . Cinzia Fionda, Giulia Malgarini, Alessandra Soriani, The journal of immunology . 2013,第12期

机译：糖原合酶激酶3的抑制作用增加NKG2D配体MICA的表达和对多发性骨髓瘤细胞NK细胞介导的细胞毒性的敏感性：STAT3的作用
2. Inhibition of glycogen synthase kinase-3 increases NKG2D ligand MICA expression and sensitivity to NK cell-mediated cytotoxicity in multiple myeloma cells: Role of STAT3 [J] . FiondaC., MalgariniG., SorianiA., The Journal of Immunology: Official Journal of the American Association of Immunologists . 2013,第12期

机译：糖原合酶激酶-3的抑制作用可增加NKG2D配体MICA的表达以及对多发性骨髓瘤细胞中NK细胞介导的细胞毒性的敏感性：STAT3的作用
3. Sodium valproate, a histone deacetylase inhibitor, augments the expression of cell-surface NKG2D ligands, MICA/B, without increasing their soluble forms to enhance susceptibility of human osteosarcoma cells to NK cell-mediated cytotoxicity. [J] . Yamanegi K, Yamane J, Kobayashi K, Oncology reports . 2010,第6期

机译：丙戊酸钠是一种组蛋白脱乙酰基酶抑制剂，可增加细胞表面NKG2D配体MICA / B的表达，而不会增加其可溶性形式来增强人骨肉瘤细胞对NK细胞介导的细胞毒性的敏感性。
4. Cancer treatment by photodynamic therapy combined with NK-cell-line-based adoptive immunotherapy [C] . Mladen Korbelik, British Columbia Cancer Agency, Vancouver, Laser-Tissue Interaction IX . 1998

机译：光动力疗法结合基于NK细胞的过继免疫疗法治疗癌症
5. Tumorigenic adenovirus 12 cells evade NK lysis by reducing the expression of NKG2D ligands [D] . Heyward, Christa Y. 2011

机译：致瘤性腺病毒12细胞通过减少NKG2D配体的表达逃避NK细胞裂解
6. Matrine increases NKG2D ligand ULBP2 in K562 cells via inhibiting JAK/STAT3 pathway: a potential mechanism underlying the immunotherapy of matrine in leukemia [O] . Xuzhang Lu, Zhichao Zhu, Lijia Jiang, 2015

机译：苦参碱通过抑制JAK / STAT3途径增加K562细胞中NKG2D配体ULBP2：苦参碱在白血病免疫治疗中的潜在机制
7. Enhanced In Vitro Expression Of NKG2D Ligands MIC A And MIC B With Histone Deacetylase Inhibitor (HDACi) Romidepsin In Pediatric Leukemia And Lymphomas: Potential for Natural Killer Cell Mediated Targeted Adoptive Cellular Immunotherapy (ACI) [O] . Satwani P., Saha A., Bavishi S., 2009

机译：组蛋白去乙酰化酶抑制剂（HDACi）罗米地辛在儿童白血病和淋巴瘤中增强了NKG2D配体MIC A和MIC B的体外表达：天然杀伤细胞介导的靶向过继细胞免疫治疗（ACI）的潜力

Romedepsin (RM), A Hdaci, Significantly Increases The Expression Of NKG2D Ligands, MIC A/B, In Leukemia/Lymphoma Cells (LL), In Part Through The Glycogen Synthase Kinase-3 (GSK-3) Pathway, Resulting In Enhanced NK Cytotoxicity: Translational Approach For Adoptive NK Cell Immunotherapy

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