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Transactivation of the EGF receptor and a PI3 kinase–ATF-1 pathway is involved in the upregulation of NOX1, a catalytic subunit of NADPH oxidase

机译：EGF受体和PI3激酶–ATF-1途径的反式激活与NOX1的上调有关，NOX1是NADPH氧化酶的催化亚基

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Fan ChunYuan; Katsuyama Masato; Nishinaka Toru; Yabe-Nishimura Chihiro;
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年度 2005
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1. 肝细胞癌中EGFR-P38 MAPK途径通过miR-675-5p上调PD-L1并通过己糖激酶2下调HL A-ABC [J] . 刘宗才, 曾春华, 王海芳, 癌症（英文版） . 2021,第004期
2. PKCδ mediates up-regulation of NOX1, a catalytic subunit of NADPH oxidase, via transactivation of the EGF receptor: possible involvement of PKCδ in vascular hypertrophy [J] . Chihiro Yabe-Nishimura, Chun?Yuan Fan, Masato Katsuyama The biochemical journal . 2005,第3期

机译：PKCδ通过EGF受体的反式激活介导NADPH氧化酶的催化亚基NOX1的上调：PKCδ可能参与血管肥大
3. PKC delta mediates up-regulation of NOX1, a catalytic subunit of NADPH oxidase, via transactivation of the EGF receptor: possible involvement of PKC delta in vascular hypertrophy [J] . Fan CY, Katsuyama M, Yabe-Nishimura C The Biochemical Journal . 2005,第3期

机译：PKC三角洲通过EGF受体的反式激活介导NADPH氧化酶的催化亚基NOX1的上调：PKC三角洲可能参与了血管肥大
4. PKCdelta mediates up-regulation of NOX1, a catalytic subunit of NADPH oxidase, via transactivation of the EGF receptor: possible involvement of PKCdelta in vascular hypertrophy. [J] . Fan CY, Katsuyama M, Yabe-Nishimura C The Biochemical Journal . 2005,第Pta3期

机译：PKCdelta通过EGF受体的反式激活介导NADPH氧化酶的催化亚基NOX1的上调：PKCdelta可能参与了血管肥大。
5. An antagonist of the prostaglandin F2alpha receptor (FP)-Galphaq-dependent response, biases FP into mitogen-activated protein kinase (MAPK) signalling through epidermal growth factor receptor (EGFR) transactivation . [D] . Wisehart, Veronica. 2011

机译：前列腺素F2alpha受体（FP）-Galphaq依赖性反应的拮抗剂通过表皮生长因子受体（EGFR）反式激活将FP偏向促分裂原激活的蛋白激酶（MAPK）信号传导。
6. PKCδ mediates up-regulation of NOX1 a catalytic subunit of NADPH oxidase via transactivation of the EGF receptor: possible involvement of PKCδ in vascular hypertrophy [O] . Chun Yuan Fan, Masato Katsuyama, Chihiro Yabe-Nishimura 2005

机译：PKCδ通过EGF受体的反式激活介导NADPH氧化酶的催化亚基NOX1的上调：PKCδ可能参与血管肥大
7. PKCδ mediates up-regulation of NOX1, a catalytic subunit of NADPH oxidase, via transactivation of the EGF receptor: possible involvement of PKCδ in vascular hypertrophy [O] . Fan, Chun Yuan, Katsuyama, Masato, Yabe-Nishimura, Chihiro 2005

机译：PKCδ通过EGF受体的反式激活介导NADPH氧化酶的催化亚基NOX1的上调：PKCδ可能参与血管肥大

Transactivation of the EGF receptor and a PI3 kinase–ATF-1 pathway is involved in the upregulation of NOX1, a catalytic subunit of NADPH oxidase

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