首页> 外文OA文献 >Triptolide, an Extracted Phytomedicine Attenuates Glomerularsclerosis in Diabetic Nephropathy Rats via Regulation of Akt/AMPK/mTOR and TGF-beta1/Smad Signaling Activities, Compared with Rapamycin

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Triptolide, an Extracted Phytomedicine Attenuates Glomerularsclerosis in Diabetic Nephropathy Rats via Regulation of Akt/AMPK/mTOR and TGF-beta1/Smad Signaling Activities, Compared with Rapamycin

机译：雷公藤甲素，一种提取的植物药可通过调节Akt / AMPK / mTOR和TGF-beta1 / Smad信号传导活性来减轻糖尿病肾病大鼠的肾小球硬化，与雷帕霉素相比

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Wan Y.G.; Mao Z.M.;
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1. Triptolide, an Extracted Phytomedicine Attenuates Glomerularsclerosis in Diabetic Nephropathy Rats via Regulation of Akt/AMPK/mTOR and TGF-beta1/Smad Signaling Activities, Compared with Rapamycin [J] . Y.G. Wan, Z.M. Mao Hong Kong Journal of Nephrology . 2015,第2期

机译：雷公藤甲素，一种提取的植物药可通过调节Akt / AMPK / mTOR和TGF-beta1 / Smad信号传导活性来减轻糖尿病肾病大鼠的肾小球硬化，与雷帕霉素相比
2. Significance of dynamic changes in gastric smooth muscle cell apoptosis, PI3K-AKT-mTOR and AMPK-mTOR signaling in a rat model of diabetic gastroparesis [J] . Zhang Mo-Han, Jiang Jing-Zhi, Cai Ying-Lan, Molecular medicine reports . 2017,第2aPta1期

机译：糖尿病胃病大鼠胃平滑肌细胞凋亡，PI3K-AKT-MTOR和AMPK-MTOR信号传导动态变化的重要性
3. The protective effects of rapamycin on cell autophagy in the renal tissues of rats with diabetic nephropathy via mTOR-S6K1-LC3II signaling pathway [J] . Lei Liu, Lijuan Yang, Baochao Chang, Renal failure. . 2018,第1期

机译：雷帕霉素通过mTOR-S6K1-LC3II信号通路对糖尿病肾病大鼠肾组织细胞自噬的保护作用
4. Tissue Kallikrein Reverses Insulin Resistance and Attenuates Nephropathy in Diabetic Rats by Activation of PI3 kinase/Akt and AMPK Signaling Pathways [O] . Gang Yuan, Juanjuan Deng, Tao Wang, -1

机译：组织激肽释放酶通过激活PI3激酶/ Akt和AMPK信号通路逆转胰岛素抵抗并减轻糖尿病大鼠的肾病
5. Triptolide, an Extracted Phytomedicine Attenuates Glomerularsclerosis in Diabetic Nephropathy Rats via Regulation of Akt/AMPK/mTOR and TGF-beta1/Smad Signaling Activities, Compared with Rapamycin [O] . Y.G. Wan, Z.M. Mao 2015

机译：雷公藤内酯醇，一种提取的植物药物通过调节akt / ampK / mTOR和TGF-β1/ smad信号活动减轻糖尿病肾病大鼠的肾小球硬化，与雷帕霉素相比较

Triptolide, an Extracted Phytomedicine Attenuates Glomerularsclerosis in Diabetic Nephropathy Rats via Regulation of Akt/AMPK/mTOR and TGF-beta1/Smad Signaling Activities, Compared with Rapamycin

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