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Slow GDP Dissociation from the guanyl nucleotide-binding site of turkey erythrocyte membranes as the limiting step in the activation of adenylate cyclase by β-adrenergic agonists

机译：从火鸡红细胞膜的鸟苷酸核苷酸结合位点缓慢释放GDP，这是β-肾上腺素能激动剂激活腺苷酸环化酶的限制步骤

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Swillens Stéphane; Juvent Michèle; Dumont Jacques E.;
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年度 1979
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1. Slow GDP Dissociation from the guanyl nucleotide‐binding site of turkey erythrocyte membranes as the limiting step in the activation of adenylate cyclase by β‐adrenergic agonists [J] . Dumont Jacques E., Juvent Michegrave, le, FEBS Letters . 1979,第2期

机译：从火鸡红细胞膜的鸟苷酸核苷酸结合位点缓慢释放GDP，这是β-肾上腺素能激动剂激活腺苷酸环化酶的限制步骤
2. Allosteric equilibrium model explains steady-state coupling of beta-adrenergic receptors to adenylate cyclase in turkey erythrocyte membranes [J] . Ugur O, Onaran HO The Biochemical Journal . 1997,第Pta3期

机译：变构平衡模型解释了火鸡红细胞膜中β-肾上腺素能受体与腺苷酸环化酶的稳态耦合
3. Allosteric equilibrium model explains steady-state coupling of β-adrenergic receptors to adenylate cyclase in turkey erythrocyte membranes [J] . H. Ongun ONARAN, ?zlem UGUR The biochemical journal . 1997,第3期

机译：变构平衡模型解释了火鸡红细胞膜中β-肾上腺素能受体与腺苷酸环化酶的稳态耦合
4. Allosteric equilibrium model explains steady-state coupling of beta-adrenergic receptors to adenylate cyclase in turkey erythrocyte membranes. [O] . O Ugur, H O Onaran 1997

机译：变构平衡模型解释了火鸡红细胞膜中β-肾上腺素能受体与腺苷酸环化酶的稳态耦合。
5. Slow GDP dissociation from the guanyl nucleotide-binding site of turkey erythrocyte membranes as the limiting step in the activation of adenylate cyclase by β-adrenergic agonists [O] . Swillens, Stéphane, Juvent, Michèle, Dumont, Jacques Emile 1979

机译：缓慢的GDP从火鸡红细胞膜的鸟苷核苷酸结合位点解离，这是β-肾上腺素能激动剂激活腺苷酸环化酶的限制步骤
6. Regulation of Postnatal beta-Adrenergic Receptor/Adenylate Cyclase Development byPrenatal Agonist Stimulation and Steroids: Alterations in Rat Kidney and Lung after Exposure to Terbutaline or Dexamethasone [R] . Kudlacz, E. M., Navarro, H. A., Kavlock, R. J., 1990

机译：通过激动剂刺激和类固醇调节出生后β-肾上腺素能受体/腺苷酸环化酶的发生：接触特布他林或地塞米松后大鼠肾和肺的改变

Slow GDP Dissociation from the guanyl nucleotide-binding site of turkey erythrocyte membranes as the limiting step in the activation of adenylate cyclase by β-adrenergic agonists

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