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An Autoimmune Disease-Associated CTLA-4 Splice Variant Lacking the B7 Binding Domain Signals Negatively in T Cells

机译：自身免疫性疾病相关的CTLA-4剪接变体缺乏T细胞中的B7结合域信号。

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Vijayakrishnan Lalitha; Slavik Jacqueline M; Illés Zsolt; Greenwald Rebecca J; Rainbow Dan; Greve Bernhard; Peterson Laurence B; Hafler David A; Freeman Gordon J; Sharpe Arlene H; Wicker Linda S; Kuchroo Vijay K;
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1. An autoimmune disease-associated CTLA-4 splice variant lacking the B7 binding domain signals negatively in T cells. [J] . Vijayakrishnan L, Slavik JM, Illes Z, Immunity . 2004,第5期

机译：自身免疫性疾病相关的CTLA-4剪接变体在T细胞中缺乏B7结合域负信号。
2. Identification of two Stat3 variants lacking a transactivation domain in grass carp: New insights into alternative splicing in the modification of teleost Stat3 signaling [J] . Fish & Shellfish Immunology . 2018,第期

机译：识别缺乏草鲤鱼中的转移结构域的两个STAT3变体：在Teleost Stat3信令的修改中替代拼接的新见解
3. A novel estrogen receptor-related protein gamma splice variant lacking a DNA binding domain exon modulates transcriptional activity of a moderate range of nuclear receptors. [J] . Kojo H, Tajima K, Fukagawa M, The Journal of Steroid Biochemistry and Molecular Biology . 2006,第4a5期

机译：缺乏DNA结合域外显子的新型雌激素受体相关蛋白γ剪接变体可调节中等范围核受体的转录活性。
4. An autoimmune disease-associated CTLA4 splice variant lacking the B7 binding domain signals negatively in T cells [C] . Lalitha Vijayakrishnan, Jacqueline M. Slavik, Zsolt Illes, Symposium on Genetics of Autoimmunity . 2005

机译：一种自身免疫疾病相关的CTLA4接头变体缺乏B7结合域在T细胞中产生负面信号
5. Novel approaches for the discovery of splice variants with cell -signaling function [D] . Leparc, German Gaston 2007

机译：发现具有细胞信号功能的剪接变体的新方法
6. An Autoimmune Disease-Associated Risk Variant in the TNFAIP3 Gene Plays a Protective Role in Brucellosis That Is Mediated by the NF-κB Signaling Pathway [O] . Lixin Lou, Wanguo Bao, Xianjun Liu, 2018

机译：TNFAIP3基因自身免疫性疾病相关风险变异在由NF-κB信号通路介导的布鲁氏菌病中起保护作用
7. Overexpression and Secretion of the Soluble CTLA-4 Splice Variant in Various Autoimmune Diseases and in Cases with Overlapping Autoimmunity [O] . Suad AlFadhli 2013

机译：各种自身免疫疾病中可溶性CTLA-4剪接变体的过度表达和分泌，以及重叠自身免疫的情况下

An Autoimmune Disease-Associated CTLA-4 Splice Variant Lacking the B7 Binding Domain Signals Negatively in T Cells

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