首页> 外文OA文献 >P2-124: Focal adhesion paxillin induces nodular cell growth, invasion, and angiogenesis in lung cancer

【2h】

P2-124: Focal adhesion paxillin induces nodular cell growth, invasion, and angiogenesis in lung cancer

机译：P2-124：局灶性黏附性Paxillin诱导肺癌中的结节性细胞生长，侵袭和血管生成

页面导航

著录项
相似文献
相关主题

著录项

作者
Jagadeeswaran Ramasamy; Zumba Osvaldo; Krishnaswamy Soundararajan; Janamanchi Varalakshmi; Seiwert Tanguy Y.; Loganathan Sivakumar; Kanteti Rajani; Nallasura Vidya; Faoro Leonardo; Vokes Everett E.; Salgia Ravi;
展开▼
作者单位

展开▼
年度 2007
总页数
原文格式 PDF
正文语种
中图分类

相似文献

外文文献
中文文献
专利

1. Hypoxia‐inducible transgelin 2 selects epithelial‐to‐mesenchymal transition and γ‐radiation‐resistant subtypes by focal adhesion kinase‐associated insulin‐like growth factor 1 receptor activation in non‐small‐cell lung cancer cells [J] . In‐Gyu Kim, Jei‐Ha Lee, Seo‐Yeon Kim, Cancer science. . 2018,第11期

机译：缺氧诱导的转运蛋白2通过非小细胞肺癌细胞中黏着斑激酶相关的胰岛素样生长因子1受体激活选择上皮向间充质转化和γ辐射抗性亚型
2. Insulin-Like Growth Factor-1 Receptor Signaling Increases the Invasive Potential of Human Epidermal Growth Factor Receptor 2-Overexpressing Breast Cancer Cells via Src-Focal Adhesion Kinase and Forkhead Box Protein M1 [J] . Sanabria-Figueroa Eduardo, Donnelly Siobhan M., Foy Kevin C., Molecular pharmacology. . 2015,第2期

机译：胰岛素样生长因子-1受体信号通过Src-Focal粘附激酶和叉头盒蛋白M1增加人表皮生长因子受体2过表达的乳腺癌细胞的侵袭潜力。
3. Syntenin increases the invasiveness of small cell lung cancer cells by activating p38, AKT, focal adhesion kinase and SP1Open [J] . Wook Youn Kim, Ji-young Jang, Yoon Kyung Jeon, Experimental & molecular medicine. . 2014,第4期

机译：Syntenin通过激活p38，AKT，粘着斑激酶和SP1Open增强小细胞肺癌细胞的侵袭性
4. Paxillin is Required for Cyclic Stretch-Induced Orientation of Endothelial Cells by Scaffolding for Accumulation of Focal Adhesion Proteins [C] . Wenjing HUANG, Naoya SAKAMOTO, Ryotaro MIYAZAWA, Mechanical Engineering Congress . 2012

机译：通过支撑局灶性粘附蛋白的积累，患有循环拉伸诱导的内皮细胞诱导取向所必需的paxillin
5. The focal adhesion protein paxillin is necessary for the activation of focal adhesion kinase. [D] . Wade, Ramon. 2003

机译：粘着斑蛋白paxillin对于激活粘着斑激酶是必需的。
6. Suppression of focal adhesion formation may account for the suppression of cell migration invasion and growth of non-small cell lung cancer cells following treatment with polyisoprenylated cysteinyl amide inhibitors [O] . Elizabeth Ntantie, Michaela J. Allen, Jerrine Fletcher, 2018

机译：用聚异戊二烯基半胱氨酰胺抑制剂治疗后抑制粘着斑形成可能是抑制非小细胞肺癌细胞迁移侵袭和生长的原因
7. Suppression of focal adhesion formation may account for the suppression of cell migration, invasion and growth of non-small cell lung cancer cells following treatment with polyisoprenylated cysteinyl amide inhibitors [O] . Elizabeth Ntantie, Michaela J. Allen, Jerrine Fletcher, 2018

机译：局灶性粘附形成的抑制可能抑制在用聚异戊二烯化的胱醇酰胺抑制剂处理后抑制非小细胞肺癌细胞的细胞迁移，侵袭和生长

P2-124: Focal adhesion paxillin induces nodular cell growth, invasion, and angiogenesis in lung cancer

著录项

相似文献

相关主题

期刊订阅