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The modulation of the rate of inactivation of the mKv1.1 K+ channel by the β subunit, Kvβ1 and lack of effect of a Kvβ1 N-terminal peptide

机译：β亚基Kvβ1对mKv1.1 K +通道失活速率的调节和Kvβ1N末端肽的作用不足

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Stephens Gary J.; Cockett Mark I.; Nawoschik S.P.; Schecter L.E.; Owen David G.;
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年度 1996
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1. The modulation of the rate of inactivation of the mKv1.1 K+ channel by the β subunit, Kvβ1 and lack of effect of a Kvβ1 N‐terminal peptide [J] . Cockett Mark I., Nawoschik S.P., Owen David G., FEBS Letters . 1996,第3期

机译：β亚基Kvβ1对mKv1.1 K +通道失活速率的调节和缺乏Kvβ1N末端肽的作用
2. The modulation of the rate of inactivation of the mKv1.1 K+ channel by the β subunit, Kvβ1 and lack of effect of a Kvβ1 N‐terminal peptide [J] . Cockett Mark I., Nawoschik S.P., Owen David G., FEBS Letters . 1996,第3期

机译：β亚基Kvβ1对mKv1.1 K +通道失活速率的调节和缺乏Kvβ1N末端肽的作用
3. Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits. [J] . Tristani-Firouzi M, Sanguinetti MC The Journal of Physiology . 1998,第Pta1期

机译：人类K +通道KvLQT1的电压依赖性失活通过与最小K +通道（minK）亚基的结合而消除。
4. Recombinant Fluorescent Ligand of Potassium Kv1.1 and Kv1.3 Channels: Design, Properties and Applications [C] . Alexey V. Feofanov, Kseniya S. Kudryashova, Anastasiya A. Ignatova, International Multidisciplinary Microscopy and Microanalysis Congress . 2017

机译：钾钾的重组荧光配体和KV1.3通道：设计，性能和应用
5. Modulation of the voltage gated potassium channel Kv4.2 by two distinct auxiliary subunits [D] . Seikel, Edward Lewis, Jr. 2008

机译：两个不同的辅助亚基对电压门控钾离子通道Kv4.2的调节
6. Fast inactivation of a brain K+ channel composed of Kv1.1 and Kvbeta1.1 subunits modulated by G protein beta gamma subunits. [O] . J Jing, D Chikvashvili, D Singer-Lahat, 1999

机译：由G蛋白βγ亚基调节的Kv1.1和Kvbeta1.1亚基组成的大脑K +通道的快速失活。
7. Voltage-dependent inactivation of the human K+ channel KvLQT1 is eliminated by association with minimal K+ channel (minK) subunits [O] . Tristani-Firouzi, Martin, Sanguinetti, Michael C 1998

机译：人类K +通道KvLQT1的电压依赖性失活通过与最小K +通道（minK）亚基的结合而消除

The modulation of the rate of inactivation of the mKv1.1 K+ channel by the β subunit, Kvβ1 and lack of effect of a Kvβ1 N-terminal peptide

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