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Suppression of late-flowering and semi-dwarf phenotypes in the arabidopsis clock mutant lhy-12;cca1-101 by phyB under continuous light

机译:连续光下phyB抑制拟南芥时钟突变体lhy-12; cca1-101中迟花和半矮表型

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摘要

Photoperiodic flowering in Arabidopsis is controlled not only by floral activators such as GI, CO and FT, but also by repressors such as SVP and FLC. Double mutations in LHY and CCA1 (lhy;cca1) accelerated flowering under short days, mainly by the GI-CO dependent pathway. In contrast, lhy;cca1 showed delayed flowering under continuous light (LL), probably due to the GI-CO independent pathway. This late-flowering phenotype was suppressed by svp, flc and elf3. However, how SVP, FLC and ELF3 mediate LHY/CCA1 and flowering time is not fully understood. We found that lhy;cca1 exhibited short hypocotyls and petioles under LL, but the molecular mechanism for these effects has not been elucidated.
机译:拟南芥中的光周期开花不仅受花卉活化剂(例如GI,CO和FT)控制,还受阻抑物(例如SVP和FLC)控制。 LHY和CCA1(lhy; cca1)的双重突变在短日内加速开花,主要是通过GI-CO依赖性途径。相比之下,lhy; cca1在连续光照(LL)下显示延迟开花,这可能是由于GI-CO独立途径引起的。 svp,flc和elf3抑制了这种迟开花表型。但是,尚未完全了解SVP,FLC和ELF3如何介导LHY / CCA1和开花时间。我们发现lhy; cca1在LL下表现出短的下胚轴和叶柄,但尚未阐明这些作用的分子机制。

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