首页> 外文OA文献 >The long non-coding RNA, GAS5, enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression

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The long non-coding RNA, GAS5, enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression

机译：长的非编码RNA GAS5通过下调IGF-1R表达来增强吉非替尼诱导的先天性EGFR酪氨酸激酶抑制剂耐药的肺腺癌细胞与广泛型EGFR的细胞死亡

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Siyuan Dong; Xiaohan Qu; Wenya Li; Xinwen Zhong; Peiwen Li; Shize Yang; Xitao Chen; Mingrui Shao; Lin Zhang;
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1. The long non-coding RNA, GAS5, enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression [J] . Siyuan Dong, Xiaohan Qu, Wenya Li, Journal of Hematology and Oncology . 2015,第1期

机译：长的非编码RNA GAS5通过下调IGF-1R表达来增强吉非替尼诱导的先天性EGFR酪氨酸激酶抑制剂耐药的肺腺癌细胞与广泛型EGFR的细胞死亡
2. The long non-coding RNA, GAS5, enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression [J] . Siyuan Dong, Xiaohan Qu, Wenya Li, Journal of Hematology and Oncology . 2015,第2期

机译：长的非编码RNA GAS5通过下调IGF-1R表达来增强吉非替尼诱导的先天性EGFR酪氨酸激酶抑制剂耐药的肺腺癌细胞与广泛型EGFR的细胞死亡
3. Expression of insulin-like growth factor 1 receptor (IGF-1R) predicts poor responses to epidermal growth factor receptor (EGFR) tyrosine kinase inhibitors in non-small cell lung cancer patients harboring activating EGFR mutations [J] . Yeo Chang Dong, Park Ki Hoon, Park Chan Kwon, Lung cancer: Journal of the International Association for the Study of Lung Cancer . 2015,第3期

机译：胰岛素样生长因子1受体（IGF-1R）的表达预测在具有激活的EGFR突变的非小细胞肺癌患者中对表皮生长因子受体（EGFR）酪氨酸激酶抑制剂的不良反应
4. Predictive Efficacy of Low Burden EGFR Mutation Detected by Next-Generation Sequencing on Response to EGFR Tyrosine Kinase Inhibitors in Non-Small-Cell Lung Carcinoma [C] . Yeul Hong Kim, Hye Sook Kim, Jae Sook Sung, Molecular Medicine TRI-CON. . 2014

机译：下一代测序对非小细胞肺癌抗蛋白酶激酶抑制剂进行下一代测序检测的低负荷EGFR突变的预测疗效
5. EGFR expression and activation in bovine cumulus cells and EGFR intramolecular regulation through interactions between tyrosines. [D] . Zhao, Zhong. 2005

机译：通过酪氨酸之间的相互作用，牛卵丘细胞中的EGFR表达和活化以及EGFR的分子内调控。
6. The long non-coding RNA GAS5 enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression [O] . Siyuan Dong, Xiaohan Qu, Wenya Li, 2015

机译：较长的非编码RNA GAS5通过下调IGF-1R表达来增强吉非替尼诱导的先天EGFR酪氨酸激酶抑制剂耐药的肺腺癌细胞与广泛型EGFR的细胞死亡
7. Microrna-144 Expression Overcomes Resistance to EGFR-Tyrosine Kinase Inhibitors in EGFR Mutant Lung Cancer Cells by Targeting Survivin [O] . Ki Eun Hwang, Eun-Taik Jeong, Hak-Ryul Kim 2016

机译：MicroRNA-144表达通过靶向Survivin克服EGFR突变体肺癌细胞EGFR-酪氨酸激酶抑制剂的抗性

The long non-coding RNA, GAS5, enhances gefitinib-induced cell death in innate EGFR tyrosine kinase inhibitor-resistant lung adenocarcinoma cells with wide-type EGFR via downregulation of the IGF-1R expression

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