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Suppression of neuroinflammation in forebrain-specific Cdk5 conditional knockout mice by PPARγ agonist improves neuronal loss and early lethality

机译：PPARγ激动剂抑制前脑特异性Cdk5条件性基因敲除小鼠的神经炎症可改善神经元丢失和早期致死率

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Elias Utreras; Ryusuke Hamada; Michaela Prochazkova; Anita Terse; Satoru Takahashi; Toshio Ohshima; Ashok B Kulkarni;
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1. Suppression of neuroinflammation in forebrain-specific Cdk5 conditional knockout mice by PPARγ agonist improves neuronal loss and early lethality [J] . Elias Utreras, Ryusuke Hamada, Michaela Prochazkova, Journal of neuroinflammation . 2014,第1期

机译：PPARγ激动剂抑制前脑特异性CDK5条件敲除小鼠的神经炎性改善了神经元丧失和早期杀伤性
2. Trans-cinnamaldehyde improves neuroinflammation-mediated NMDA receptor dysfunction and memory deficits through blocking NF-kappa B pathway in presenilin1/2 conditional double knockout mice [J] . Zhao Yang, Deng Haiyan, Li Kun, Brain, Behavior, and Immunity . 2019,第1期

机译：Trans-cinnamaldehyde通过阻塞Presenilin1 / 2条件双敲除小鼠阻断NF-Kappa B途径改善神经炎性介导的NMDA受体功能障碍和记忆缺陷
3. Xenon improves long-term cognitive function, reduces neuronal loss and chronic neuroinflammation, and improves survival after traumatic brain injury in mice [J] . Campos-Pires Rita, Hirnet Tobias, Valeo Flavia, British journal of anaesthesia . 2019,第1期

机译：氙气改善了长期的认知功能，减少了神经元损失和慢性神经炎症，并在小鼠创伤性脑损伤后提高存活
4. Self-Expanding, In Situ Forming, Polyurethane Foam Reduces Blood Loss And Improves Survival In A Lethal, Battlefield Injury Model in Swine [C] . Upma Sharma, Greg Zugates, Adam Rago, Society for Biomaterials fall symposium 2012.;vol. 34. . 2012

机译：自膨胀，原位形成的聚氨酯泡沫在猪的致命战场伤害模型中减少失血并提高生存率
5. Characterization of novel Col5a3 knockout and Tll1 conditional knockout mice. [D] . Butz, Delana Hopkins. 2009

机译：新型Col5a3基因敲除和Tll1条件基因敲除小鼠的表征。
6. Suppression of neuroinflammation in forebrain-specific Cdk5 conditional knockout mice by PPARγ agonist improves neuronal loss and early lethality [O] . Elias Utreras, Ryusuke Hamada, Michaela Prochazkova, 2014

机译：PPARγ激动剂抑制前脑特异性Cdk5条件性基因敲除小鼠的神经炎症可改善神经元丢失和早期致死率
7. A novel non-agonist peroxisome proliferator-activated receptor γ (PPARγ) ligand UHC1 blocks PPARγ phosphorylation by cyclin-dependent kinase 5 (CDK5) and improves insulin sensitivity [O] . Choi, Sun-Sil, Kim, Eun Sun, Koh, Minseob, 2014

机译：一种新型的非激动剂过氧化物酶体增殖物激活受体γ（PPARγ）配体UHC1通过细胞周期蛋白依赖性激酶5（CDK5）阻止PPARγ磷酸化，并提高胰岛素敏感性

Suppression of neuroinflammation in forebrain-specific Cdk5 conditional knockout mice by PPARγ agonist improves neuronal loss and early lethality

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