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Obesity enhances nongenomic estrogen receptor crosstalk with the PI3K/Akt and MAPK pathways to promote measures of breast cancer progression

机译：肥胖通过PI3K / Akt和MAPK途径增强非基因组雌激素受体的串扰，从而促进乳腺癌进展的评估

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1. Obesity enhances nongenomic estrogen receptor crosstalk with the PI3K/Akt and MAPK pathways to promote in vitro measures of breast cancer progression [J] . Laura W Bowers, David A Cavazos, Ilane XF Maximo, Breast Cancer Research . 2013,第4期

机译：肥胖通过PI3K / Akt和MAPK途径增强非基因组雌激素受体的串扰，从而促进乳腺癌进展的体外测量
2. Spatholobus suberectus Column Extract Inhibits Estrogen Receptor Positive Breast Cancer via Suppressing ER MAPK PI3K/AKT Pathway [J] . Jia-Qi Sun, Gan-Lin Zhang, Yi Zhang, Evidence-based complementary and alternative medicine: eCAM . 2016,第11期

机译：Spatholobus suberectus柱提取物通过抑制ER Mapk Pi3k / Akt途径抑制雌激素受体阳性乳腺癌
3. Microarray phosphatome profiling of breast cancer patients unveils a complex phosphatase regulatory role of the MAPK and PI3K pathways in estrogen receptor-negative breast cancers [J] . Ramon G. Manzano, Elena M. Martinez-Navarro, Jeronimo Forteza, International journal of oncology . 2014,第6期

机译：乳腺癌患者的微阵列磷原子分析揭示了雌激素受体阴性乳腺癌中MAPK和PI3K途径的复杂磷酸酶调节作用
4. Ang-2 promoting proliferation of SW1116 colon cancer cells through PI3K/Akt pathways [C] . Hong Zhang Ji, Yang Wen Chun, Ping Wang Ai, 2011 International Conference on Human Health and Biomedical Engineering . 2011

机译：Ang-2通过PI3K / Akt途径促进SW1116结肠癌细胞的增殖
5. DLX4 homeoprotein promotes PI3K/Akt anti-apoptosis pathway in ER negative breast cancer cells through upregulation of VEGFA. [D] . Wu, Weiwei. 2010

机译：DLX4同源蛋白通过上调VEGFA促进ER阴性乳腺癌细胞中的PI3K / Akt抗凋亡途径。
6. Obesity enhances nongenomic estrogen receptor crosstalk with the PI3K/Akt and MAPK pathways to promote in vitro measures of breast cancer progression [O] . Laura W Bowers, David A Cavazos, Ilane XF Maximo, 2013

机译：肥胖通过PI3K / Akt和MAPK途径增强非基因组雌激素受体的串扰以促进乳腺癌进展的体外测量
7. The Akt/mTOR Pathway and Estrogen Receptor Phosphorylations : a crosstalk with potential to predict tamoxifen resistance in breast cancer [O] . Bostner, Josefine 2013

机译：Akt / mTOR途径和雌激素受体磷酸化：串扰有可能预测乳腺癌中他莫昔芬的耐药性

Obesity enhances nongenomic estrogen receptor crosstalk with the PI3K/Akt and MAPK pathways to promote measures of breast cancer progression

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