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Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis

机译：温阳化浊通络配方，通过下调全身性硬化症中survivin和cyclin D1的表达，促进细胞凋亡和抑制增殖，从而改善皮肤纤维化

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Li Han; Hua Bian; Jingfeng Ouyang; Yuefeng Bi; Lei Yang; Songshan Ye;
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1. Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis [J] . Li Han, Hua Bian, Jingfeng Ouyang, BMC Complementary and Alternative Medicine . 2016,第1期

机译：温阳化浊通络配方，通过下调系统性硬化症中survivin和cyclin D1的表达，促进细胞凋亡和抑制增殖，从而改善皮肤纤维化
2. Wenyang Huazhuo Tongluo formula inhibits fibrosis via suppressing Wnt/β-catenin signaling pathway in a Bleomycin-induced systemic sclerosis mouse model [J] . Qian Wang, Wenhua Zang, Li Han, Chinese Medicine . 2018,第1期

机译：文阳化浊通络配方通过抑制博来霉素诱导的系统性硬化小鼠模型中的Wnt /β-catenin信号传导途径来抑制纤维化
3. Yifei Xuanfei Jiangzhuo formula, a Chinese herbal decoction, improves memory impairment through inhibiting apoptosis and enhancing PKA/CREB signal transduction in rats with cerebral ischemia/reperfusion [J] . Wu Lin, Zhao Qing-Shan, Li Tian-Wei, Molecular medicine reports . 2015,第3aPta2期

机译：中药益肺宣肺降浊方通过抑制脑缺血/再灌注大鼠的细胞凋亡和增强PKA / CREB信号转导来改善记忆障碍
4. Wenyang Huazhuo Tongluo formula a Chinese herbal decoction improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis [O] . Li Han, Hua Bian, Jingfeng Ouyang, 2016

机译：温阳化浊通络配方通过下调系统性硬化症中survivin和cyclin D1的表达促进细胞凋亡并抑制增殖从而改善皮肤纤维化
5. Wenyang Huazhuo Tongluo formula inhibits fibrosis via suppressing Wnt/β-catenin signaling pathway in a Bleomycin-induced systemic sclerosis mouse model [O] . Qian Wang, Wenhua Zang, Li Han, 2018

机译：温阳华泽通络惯用型通过抑制玻璃霉素诱导的全身硬化小鼠模型中的Wnt /β-catenin信号传导途径抑制纤维化

Wenyang Huazhuo Tongluo formula, a Chinese herbal decoction, improves skin fibrosis by promoting apoptosis and inhibiting proliferation through down-regulation of survivin and cyclin D1 in systemic sclerosis

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