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Moniliformediquinone as a potential therapeutic agent, inactivation of hepatic stellate cell and inhibition of liver fibrosis in vivo

机译：Moniliformediquinone作为潜在的治疗剂，可在体内灭活肝星状细胞并抑制肝纤维化

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Tsui-Hwa Tseng; Wea-Lung Lin; Zi-Hui Chen; Yean-Jang Lee; Ming-Shiun Shie; Kam-Fai Lee; Chien-Heng Shen; Hsing-Chun Kuo;
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1. Moniliformediquinone as a potential therapeutic agent, inactivation of hepatic stellate cell and inhibition of liver fibrosis in vivo [J] . Tsui-Hwa Tseng, Wea-Lung Lin, Zi-Hui Chen, Journal of Translational Medicine . 2016,第1期

机译：Moniliformediquinone作为潜在的治疗剂，可在体内灭活肝星状细胞并抑制肝纤维化
2. STAT3 inhibition suppresses hepatic stellate cell fibrogenesis: HJC0123, a potential therapeutic agent for liver fibrosis [J] . Lopez Omar Nunez, Bohanon Fredrick J., Wang Xiaofu, RSC Advances . 2016,第102期

机译：STAT3抑制抑制肝星状细胞纤维发生：HJC0123，肝纤维化的潜在治疗剂
3. Inhibition of ASICs reduces rat hepatic stellate cells activity and liver fibrosis: an in vitro and in vivo study. [J] . Chun-xiao Pan, Fan-rong Wu, Xiao-yu Wang, Cell biology international. . 2014,第9期

机译：抑制ASIC会降低大鼠肝星状细胞活性和肝纤维化：一项体外和体内研究。
4. MicroRNA-506-3p Regulated the Proliferation of Hepatic Stellate Cells during Liver Fibrosis [C] . Junhua Huang, Yang Ao, Hao Li International Conference on Frontiers of Biological Sciences and Engineering . 2020

机译：MicroRNA-506-3P在肝纤维化期间调节肝星状细胞的增殖
5. Emerging Roles of Platelet-Derived Growth Factor Receptor Alpha in Chronic Liver Injury: Potential Therapeutic Target in Hepatic Fibrosis [D] . Kikuchi, Alexander Thomas. 2017

机译：血小板源性生长因子受体α在慢性肝损伤中的新兴作用：肝纤维化的潜在治疗靶点。
6. Moniliformediquinone as a potential therapeutic agent inactivation of hepatic stellate cell and inhibition of liver fibrosis in vivo [O] . Tsui-Hwa Tseng, Wea-Lung Lin, Zi-Hui Chen, 2016

机译：Moniliformediquinone作为潜在的治疗剂可在体内灭活肝星状细胞并抑制肝纤维化
7. STAT3 inhibition suppresses hepatic stellate cell fibrogenesis: HJC0123, a potential therapeutic agent for liver fibrosis [O] . Omar Nunez Lopez, Fredrick J. Bohanon, Xiaofu Wang, 2016

机译：STAT3抑制抑制肝星状细胞纤维发生：HJC0123，肝纤维化的潜在治疗剂

Moniliformediquinone as a potential therapeutic agent, inactivation of hepatic stellate cell and inhibition of liver fibrosis in vivo

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