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〈Originals〉Relationship between Blood Lactate Concentration and Excessive CO_2 Expiration During and after Ramp Exercise

机译:〈原始〉坡道运动期间和之后血乳酸浓度与过量CO_2排放之间的关系

摘要

YUNOKI, T., OGATA, H. and YANO, T., Relationship between Blood Lactate Concentration and Excessive CO_2 Expiration During and after Ramp Exercise. Abv. Exerc. Sports Physiol., Vol.9, No.3 pp.97-103, 2003. The purpose of this study was to examine the kinetics of excessively expired CO2 and the blood lactate concentration ([La-]) during and after ramp exercise. Six healthy males performed three different ramp exercise tests in which the work rate was increased as a ramp function at 10 W/min (R10), 20 W/min (R20) and 40 W/min (R40). Excessive CO2 expiration during exercise (CO2 excess) significantly correlated with the [La-] increase during exercise (△[La-]). Excessive CO2 expiration during recovery (post CO2 excess) significantly correlated with the [La-] increase during recovery (post △[La-]). However, Co2 excess in R40 was significantly lower compared with R20 and R10, despite there being no significant difference in △[La-] among the three tests, In addition, post CO2 excess showed a positive value even when post △[La-] was zero. Endtidal CO2 pressure (PETCO2) decreased from the onset of respiratory compensation to about 10 min post-exercise in all tests. PETCO2 decrement during exercise tended to be greater in R10 than in R40, and PETCO2 decrement during post-exercise tended to be greater in R40 than in R10. OETCO2 was significantly related to [La-] during both at exhaustion and at 10 min post-exercise. However, PETCO2 at the same [La-] was lower at 10 min post-exercise than at exhaustion. Furthermore, alactic O2 debt, which was calulated from O2 uptake kinetics during recovery, was significantly higher in R40 than in the other tests. These results suggest that there is a case in whichi the blood lactate is not always the couse of excessive CO2 expriration in response to ramp exercise. The decrease in PETCO2 and the H+ uptake-release by breakdown-resynthesis of phosphocreatine were thought to be main factors influencing the excessive CO2 expiration as well as the [La-} increase.
机译:田野由纪纪,绪方贺田和矢野,O野,运动期间及之后血乳酸浓度与过量CO_2呼出之间的关系。绝对值锻炼运动生理学》,第9卷,第3期,第97-103页,2003年。本研究的目的是检查斜向运动期间和运动后过度呼出的CO2的动力学和血液乳酸浓度([La-])。六名健康的男性进行了三种不同的坡道运动测试,其中工作率随着坡道功能的增加而增加,分别为10 W / min(R10),20 W / min(R20)和40 W / min(R40)。运动过程中二氧化碳过多(CO2过量)与运动过程中[La-]增加(△[La-])显着相关。恢复过程中过量的CO2排放(后CO2过量)与恢复过程中的[La-]增加(后Δ[La-])显着相关。但是,尽管三个测试之间的△[La-]没有显着差异,但R40中的CO2过量明显低于R20和R10。此外,即使在△[La-]之后,CO2过量也显示为正值。是零。在所有测试中,呼气末二氧化碳压力(PETCO2)从呼吸补偿开始降低到运动后约10分钟。 R10的运动过程中的PETCO2减量趋于大于R40,R40的运动后的PETCO2的减量趋于大于R10。在疲惫和运动后10分钟,OETCO2与[La-]均显着相关。但是,在运动后10分钟,相同的[La-]下的PETCO2低于疲惫。此外,根据恢复过程中的O2吸收动力学计算得出的Al2O2负债在R40中显着高于其他测试。这些结果表明,在某些情况下,血液乳酸并不总是因过度锻炼而引起过多的CO2释放。磷酸肌酸的分解-再合成引起的PETCO2的减少和H +的吸收释放被认为是影响CO2过度释放以及[La-}增加的主要因素。

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