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Dentin hypersensitivity: pain mechanisms and aetiology of exposed cervical dentin

机译:牙本质过敏:暴露的子宫颈牙本质的疼痛机制和病因

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摘要

OBJECTIVESududThe paper's aim is to review dentin hypersensitivity (DHS), discussing pain mechanisms and aetiology.ududMATERIALS AND METHODSududLiterature was reviewed using search engines with MESH terms, DH pain mechanisms and aetiology (including abrasion, erosion and periodontal disease).ududRESULTSududThe many hypotheses proposed for DHS attest to our lack of knowledge in understanding neurophysiologic mechanisms, the most widely accepted being the hydrodynamic theory. Dentin tubules must be patent from the oral environment to the pulp. Dentin exposure, usually at the cervical margin, is due to a variety of processes involving gingival recession or loss of enamel, predisposing factors being periodontal disease and treatment, limited alveolar bone, thin biotype, erosion and abrasion.ududCONCLUSIONSududThe current pain mechanism of DHS is thought to be the hydrodynamic theory. The initiation and progression of DHS are influenced by characteristics of the teeth and periodontium as well as the oral environment and external influences. Risk factors are numerous often acting synergistically and always influenced by individual susceptibility.ududCLINICAL RELEVANCEududWhilst the pain mechanism of DHS is not well understood, clinicians need to be mindful of the aetiology and risk factors in order to manage patients' pain and expectations and prevent further dentin exposure with subsequent sensitivity.
机译:目的 ud ud本文旨在综述牙本质过敏症(DHS),讨论疼痛机制和病因。 ud ud材料和方法 ud ud文献使用具有MESH术语,DH疼痛机制和病因(包括磨损,侵蚀和牙周疾病)。 ud ud结果 ud ud针对DHS提出的许多假设证明我们缺乏对神经生理机制的了解,最广泛接受的是流体力学理论。从口腔环境到牙髓,牙本质小管必须具有专利权。牙本质暴露通常发生在宫颈边缘,归因于各种过程,包括牙龈退缩或牙釉质丧失,易感因素包括牙周疾病和治疗,牙槽骨受限,生物型薄,侵蚀和磨损。 ud ud结论 ud udDHS当前的疼痛机制被认为是流体力学理论。 DHS的启动和发展受牙齿和牙周膜的特性以及口腔环境和外部影响的影响。危险因素通常协同作用,并且总是受到个体敏感性的影响。 ud ud临床相关性 ud ud尽管对DHS的疼痛机制尚不十分了解,但临床医生需要注意病因和危险因素,以便管理患者的病情。疼痛和期望,并防止随后的敏感性进一步暴露于牙本质。

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