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Histone deacetylase inhibitors decrease NHEJ both by acetylation of repair factors and trapping of PARP1 at DNA double-strand breaks in chromatin

机译：组蛋白脱乙酰酶抑制剂通过乙酰化因子乙酰化和染色质DNA双链突破的PARP1捕获而降低NHEJ

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Carine Robert; Pratik K. Nagaria; Nisha Pawar; Adeoluwa Adewuyi; Ivana Gojo; David J. Meyers; Philip A. Cole; Feyruz V. Rassool;
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1. Histone Deacetylase Inhibitors Sensitize Prostate Cancer Cells to Agents that Produce DNA Double-Strand Breaks by Targeting Ku70 Acetylation (Retraction of Vol 67, Pg 5318, 2007) [J] . Kulp Samuel K., Matsuyama Shigemi, Chen Ching-Shih, Cancer research: The official organ of the American Association for Cancer Research, Inc . 2018,第14期

机译：组蛋白脱乙酰化酶抑制剂通过靶向Ku70乙酰化（R-67，PG 5318,2007）来敏化前列腺癌细胞对产生DNA双链断裂的药剂的药物
2. Histone H3K56 Acetylation, Rad52, and Non-DNA Repair Factors Control Double-Strand Break Repair Choice with the Sister Chromatid [J] . Sandra Mu?oz-Galván, Sonia Jimeno, Rodney Rothstein, PLoS Genetics . 2013,第1期

机译：组蛋白H3K56乙酰化，RAD52和非DNA修复因子控制双链脱离修复选择与姐妹染色体
3. E2F1 acetylation directs p300/CBP-mediated histone acetylation at DNA double-strand breaks to facilitate repair [J] . Swarnalatha Manickavinayaham, Renier Vélez-Cruz, Anup K. Biswas, Nature Communications . 2019,第1期

机译：E2F1乙酰化直接P300 / CBP介导的组蛋白乙酰化在DNA双链突破以便于修复
4. Spatial and Temporal Organisation of Multiprotein Systems of Cell Regulation and Signalling: What Can We Learn from NHEJ System of Double-Strand Break Repair? [C] . Qian Wu, Lynn Sibanda, Takashi Ochi, Macromolecular crystallography: deciphering the structure, function and dynamics of biological molecules . 2010

机译：细胞调节和信号传导多蛋白系统的时空组织：我们可以从NHEJ双链断裂修复系统中学到什么？
5. Computational Modeling of Chromatin Organization and Histone Phosphorylation during the Repair of DNA Double-Strand Breaks [D] . Bronk, Gabriel 2019

机译：DNA双链断裂修复过程中染色质组织和组蛋白磷酸化的计算模型。
6. Histone deacetylase inhibitors decrease NHEJ both by acetylation of repair factors and trapping of PARP1 at DNA double-strand breaks in chromatin [O] . Carine Robert, Pratik K. Nagaria, Nisha Pawar, -1

机译：组蛋白脱乙酰基酶抑制剂通过修复因子的乙酰化和PARP1在染色质的DNA双链断裂处的捕获而降低NHEJ
7. Histone deacetylase inhibitors sensitize prostate cancer cells to agents that produce DNA double-strand breaks by targeting Ku70 acetylation [O] . Chen, CS 2008

机译：组蛋白脱乙酰基酶抑制剂通过靶向Ku70乙酰化作用使前列腺癌细胞对产生DNA双链断裂的药物敏感

Histone deacetylase inhibitors decrease NHEJ both by acetylation of repair factors and trapping of PARP1 at DNA double-strand breaks in chromatin

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